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However, it acquired an important practical use with the advent of serum therapy to treat certain types of pneumococcal pneumonia in the 1920s because selection of the proper antiserum to treat an individual patient required correct identification of the infecting pneumococcal serotype, and the quellung reaction was the only method available to ...
S.pneumoniae growth on blood agar. Streptococcus pneumoniae can be differentiated from the viridans streptococci, some of which are also alpha-hemolytic, using an optochin test, as S. pneumoniae is optochin-sensitive. S. pneumoniae can also be distinguished based on its sensitivity to lysis by bile, the so-called "bile
Streptococcus pseudopneumoniae is a gram-positive coccus that may cause pneumonia in humans. [1] [2] It was first described in 2004. [1] The organism is often mistaken for S. pneumoniae [1] and its clinical importance is as yet uncertain. It seems likely that most cases of S. pseudopneumoniae pneumonia are misdiagnosed as S. pneumoniae. [2]
Antibiotic resistance tests: Bacteria are streaked on dishes with white disks, each impregnated with a different antibiotic. Clear rings, such as those on the left, show that bacteria have not grown—indicating that these bacteria are not resistant. The bacteria on the right are fully resistant to all but two of the seven antibiotics tested. [33]
Fecal occult blood testing (FOBT), as its name implies, aims to detect subtle blood loss in the gastrointestinal tract, anywhere from the mouth to the colon.Positive tests ("positive stool") may result from either upper gastrointestinal bleeding or lower gastrointestinal bleeding and warrant further investigation for peptic ulcers or a malignancy (such as colorectal cancer or gastric cancer).
At baseline, walking pneumonia is a respiratory tract infection, causing illnesses that can range from mild to severe, per the CDC. It can take one to four weeks to develop symptoms of walking ...
Further identification can be done serologically to test for the presence of capsular antigen, which is the dominant structure on the surface of S. pneumoniae. [2] [5] Bile solubility can be used to further distinguish S. pneumoniae from viridans streptococci as S. pneumoniae are bile soluble and viridans streptococci are not. [6]
S. pneumoniae, S. mitis and S. oralis can become competent, and as a result actively acquire homologous DNA for transformation by a predatory fratricidal mechanism [42] This fratricidal mechanism mainly exploits non-competent siblings present in the same niche [43] Among highly competent isolates of S. pneumoniae, Li et al. [44] showed that ...