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Hypoplastic left heart syndrome (HLHS) is a rare congenital heart defect in which the left side of the heart is severely underdeveloped and incapable of supporting the systemic circulation. [2] It is estimated to account for 2-3% of all congenital heart disease. [ 3 ]
Hypoplastic left heart syndrome, or HLHS, is a congenital heart defect that affects the left side of the heart, specifically affecting the left ventricle and ascending aorta. Also, the aortic valve and mitral valve might be either too small to allow enough blood to flow through, or they might be absent altogether, called atresia.
Norwood procedure is most commonly performed to treat hypoplastic left heart syndrome, double outlet right ventricle, double inlet left ventricle, and other single ventricle congenital heart defects. [5] Variations are also used for palliation of mitral and tricuspid atresia [6] and subsets of transposition of great arteries (TGA). [7]
4-year-old who received new heart after waiting 1,025 days goes home from hospital. According to Cleveland Clinic Children's, John-Henry was born with hypoplastic left heart syndrome, or HLHS, a ...
From days 23 through 28, the heart tube folds and twists, with the future ventricles moving left of center (the ultimate location of the heart) and the atria moving towards the head. [ 39 ] On day 28, areas of tissue in the heart tube begin to expand inwards; after about two weeks, these expansions (the membranous " septum primum " and the ...
The Fontan Kreutzer procedure is used in pediatric patients who possess only a single functional ventricle, either due to lack of a heart valve (e.g. tricuspid or mitral atresia), an abnormality of the pumping ability of the heart (e.g. hypoplastic left heart syndrome or hypoplastic right heart syndrome), or a complex congenital heart disease where a bi-ventricular repair is impossible or ...
Hypoplastic Left Heart Syndrome (HLHS) it is treated with the Norwood procedure. This typically consists of three surgeries creating and removing shunts . The atrial septum is removed, the aortic arch is reconstructed to remove any hypoplasia , and then the main pulmonary artery is connected into this reconstructed arch, resulting in the right ...
The classical triad of symptoms that defines 3C syndrome includes certain heart defects, hypoplasia (underdevelopment) of the cerebellum, and cranial dysmorphisms, which can take various forms. The heart defects and cranial dysmorphisms are heterogeneous in individuals who are all classed as having Ritscher-Schinzel syndrome. [2]