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Examples are deep vein thrombosis and pulmonary embolism, the risk of which can be mitigated by certain interventions, such as the administration of anticoagulants (e.g., warfarin or low molecular weight heparins), antiplatelet drugs (e.g., aspirin), compression stockings, and cyclical pneumatic calf compression in high risk patients.
Blood is drained from the venous (deoxygenated) circulation, and is cycled through the CPB machine. While in the machine, the blood is filtered, heated or cooled, and infused with oxygen. Subsequently, it is pumped back into the arterial (oxygenated) circulation, thereby bypassing the heart and lungs and maintaining the perfusion of the vital ...
There is some risk of damage to the pleural cavities around the lungs, which can lead to pneumonia, or pleural effusion. [2] It also presents typical surgical risks, such as infection, anaesthesia complications, blood clots, and bleeding. [2] [3] There is a low risk of haemorrhage if the heart is perforated whilst removing the pericardium. [3]
Iatrogenic hemothorax can occur as a complication of heart and lung surgery, for example the rupture of lung arteries caused by the placement of catheters, thoracotomy, thoracostomy, or thoracentesis. The most common iatrogenic causes include subclavian venous catheterizations and chest tube placements, with an occurrence rate of around 1%. [5]
A lung illustration depicting a pulmonary embolism as a thrombus (blood clot) that has travelled from another region of the body, causes occlusion of the pulmonary bronchial artery, leading to arterial thrombosis of the superior and inferior lobes in the left lung: Specialty: Hematology, cardiology, pulmonology, Emergency medicine: Symptoms
Surgery is indicated in patients with pulmonary artery emboli that are surgically accessible. Thrombi are usually the cause of recurrent/chronic pulmonary emboli and therefore of chronic thromboembolic pulmonary hypertension (CTEPH). [2] PTE is the only definitive treatment option available for CTEPH. [3]
The pathophysiology of pulmonary heart disease (cor pulmonale) has always indicated that an increase in right ventricular afterload causes RV failure (pulmonary vasoconstriction, anatomic disruption/pulmonary vascular bed and increased blood viscosity are usually involved [1]), however most of the time, the right ventricle adjusts to an overload in chronic pressure.
Chronic thromboembolic pulmonary hypertension (CTEPH) is a long-term disease caused by a blockage in the blood vessels that deliver blood from the heart to the lungs (the pulmonary arterial tree). These blockages cause increased resistance to flow in the pulmonary arterial tree which in turn leads to rise in pressure in these arteries ...
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