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Terms oligospermia, oligozoospermia, and low sperm count refer to semen with a low concentration of sperm [1] and is a common finding in male infertility. Often semen with a decreased sperm concentration may also show significant abnormalities in sperm morphology and motility (technically oligoasthenoteratozoospermia). There has been interest ...
The volume of the semen sample (must be more than 1.5 ml), approximate number of total sperm cells, sperm motility/forward progression, and % of sperm with normal morphology are measured. It is possible to have hyperspermia (high volume more than 6 ml) or Hypospermia (low volume less than 0.5 ml). This is the most common type of fertility testing.
Collecting the sperm at home gives a higher sperm concentration, sperm count and motility, particularly if the sperm is collected via sexual intercourse. [35] If the semen sample is to be collected by masturbation, a specimen from the early stages of the ejaculate should be into a clean, unused, sealed collection cup.
Sperm count is determined with a simple test called a semen analysis that looks at sperm concentration, or how many sperm there are per milliliter of fluid. There should be about 100,000,000 sperm ...
Total motile spermatozoa (TMS) [25] or total motile sperm count (TMSC) [26] is a combination of sperm count, motility and volume, measuring how many million sperm cells in an entire ejaculate are motile. Use of approximately 20 million sperm of motility grade c or d in ICI, and 5 million ones in IUI may be an approximate recommendation.
Various environmental and lifestyle factors have been proposed to explain the decline in semen quality observed over the last 50 years.
In both men and women, ASA production are directed against surface antigens on sperm, which can interfere with sperm motility and transport through the female reproductive tract, inhibiting capacitation and acrosome reaction, impaired fertilization, influence on the implantation process, and impaired growth and development of the embryo. The ...
Sperm motility is dependent on several metabolic pathways and regulatory mechanisms. The axonemal bend movement is based on the active sliding of axonemal doublet microtubules by the molecular motor dynein, which is divided into an outer and an inner arm. Outer and inner arm plays different roles in the production and regulation of flagellar motility: the outer arm increase the bea