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There is a substantial amount of empirical research on negative affect (NA) and its role in the tripartite model. For example, the Mood and Anxiety Symptom Questionnaire (MASQ) [ 10 ] was administered to a sample of college students and a sample of psychiatric patients.
The largest limitation of evolutionary explanations of depression, which include rank theory, is the lack of falsifiability. [9] While these theories provide "reasonably parsimonious" explanations, [10] they are not grounded in empirical research, which severely affects their real-world application.
Depressive realism is the hypothesis developed by Lauren Alloy and Lyn Yvonne Abramson [1] that depressed individuals make more realistic inferences than non-depressed individuals.
Depression is a significant mental illness with physiological and psychological consequences, including sluggishness, diminished interest and pleasure, and disturbances in sleep and appetite. [1] It is predicted that by the year 2030, depression will be the number one cause of disability in the United States and other high-income countries. [2]
It is an official journal of the Anxiety and Depression Association of America and covers research on depressive and anxiety disorders. The editor-in-chief as of July 1, 2017 is Murray B. Stein ( University of California, San Diego ).
The biology of depression is the attempt to identify a biochemical origin of depression, as opposed to theories that emphasize psychological or situational causes. Scientific studies have found that different brain areas show altered activity in humans with major depressive disorder (MDD) . [ 1 ]
Instead, depression was assessed using an ex-nihilo study scale (QIDS-SR), which was used for both medical decision-making and scientific evaluation. [ 8 ] STAR*D changed the inclusion and exclusion criteria for subjects during the study, so 931 subjects were included when they met the exclusion criteria, and 370 subjects were excluded while ...
The neurogenesis hypothesis of depression gained popularity due to the large number of correlative studies which indicate a relationship between impaired hippocampal neurogenesis and depression. For example, reduced hippocampal volume is associated with depression, which impaired neurogenesis may contribute to. [6] Conflicting evidence