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During development, blood vessels grow from the central part of the retina outwards. This process is completed a few weeks before the normal time of delivery. However, in premature babies, the process has yet to be completed. If the vessels grow and branch abnormally, the baby becomes susceptible to developing ROP.
The muscles of the eye such as ciliary muscles – become stronger after two months of age, allowing infants to focus on particular objects through contraction and relaxation. Their retinal images are also smaller compared to adults due to shorter distances from the retina to the cornea of the infants' eye. [1]
Congenital blindness is a hereditary disease and can be treated by gene therapy. Visual loss in children or infants can occur either at the prenatal stage (during the time of conception or intrauterine period) or postnatal stage (immediately after birth). [3] There are multiple possible causes of congenital blindness.
Many transcription factors for eye development are also involved in the morphogenesis of forebrain, which may explain why ONH is commonly a part of a syndrome involving brain malformations. [ 2 ] ONH impacts all ethnic groups, although in the United States, occurrence is lower in persons of Asian descent.
Cyclopia (named after the Greek mythology characters cyclopes), also known as alobar holoprosencephaly, is the most extreme form of holoprosencephaly and is a congenital disorder (birth defect) characterized by the failure of the embryonic prosencephalon to properly divide the orbits of the eye into two cavities.
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Eye development is initiated by the master control gene PAX6, a homeobox gene with known homologues in humans (aniridia), mice (small eye), and Drosophila (eyeless). The PAX6 gene locus is a transcription factor for the various genes and growth factors involved in eye formation. [1] [5] Eye morphogenesis begins with the evagination, or ...
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