Search results
Results from the WOW.Com Content Network
Focal and diffuse brain injury are ways to classify brain injury: focal injury occurs in a specific location, while diffuse injury occurs over a more widespread area. It is common for both focal and diffuse damage to occur as a result of the same event; many traumatic brain injuries have aspects of both focal and diffuse injury. [ 1 ]
Focal neurological deficits may be caused by a variety of medical conditions such as head trauma, [1] tumors or stroke; or by various diseases such as meningitis or encephalitis or as a side effect of certain medications such as those used in anesthesia. [2] Neurological soft signs are a group of non-focal neurologic signs. [3]
Headaches and pain can occur as a result of a brain injury, either directly from the damage or due to neurological conditions stemming from the injury. Due to the changes in the brain as well as the issues associated with the change in physical and mental capacity, depression and low self-esteem are common side effects that can be treated with ...
Brain ischemia has been linked to a variety of diseases or abnormalities. Individuals with sickle cell anemia, compressed blood vessels, ventricular tachycardia, plaque buildup in the arteries, blood clots, extremely low blood pressure as a result of heart attack, and congenital heart defects have a higher predisposition to brain ischemia in comparison to the average population.
Early myoclonic epileptic encephalopathy (possibly due to metabolic disorders). Gluten encephalopathy: Focal abnormalities of the white matter (generally area of low perfusion) are appreciated through magnetic resonance. Migraine is the most common symptom reported.
Treatment Endovascular coiling , surgical clipping , cerebral bypass surgery, pipeline embolization An intracranial aneurysm , also known as a cerebral aneurysm , is a cerebrovascular disorder characterized by a localized dilation or ballooning of a blood vessel in the brain due to a weakness in the vessel wall.
Risks for late PTS include hydrocephalus, reduced blood flow to the temporal lobes of the brain, [1] brain contusions, subdural hematomas, [5] a torn dura mater, and focal neurological deficits. [9] PTA that lasts for longer than 24 hours after the injury is a risk factor for both early and late PTS. [1]
focal-neurological deficits; Elevated titres of thyroid tissue antibodies (TPO-ab or microsomal) Euthyroidism (potentially achieved by treatment with L-T4 or L-T3) or mild hypothyroidism with TSH concentration below 20 mIU/L; No evidence for infectious, toxic, metabolic or neoplastic processes in blood, urine or CSF analyses