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The biology of depression is the attempt to identify a biochemical origin of depression, as opposed to theories that emphasize psychological or situational causes. Scientific studies have found that different brain areas show altered activity in humans with major depressive disorder (MDD) . [ 1 ]
One reason depression is thought to be a pathology is that it causes so much psychic pain and distress. However, physical pain is also very distressful, yet it has an evolved function: to inform the organism that it is being damaged, to motivate it to withdraw from the source of damage, and to learn to avoid such damage-causing circumstances in the future.
Syphilis; the prevalence of which is on the rise, is another of the "great imitators", which if left untreated can progress to neurosyphilis and affect the brain, can present with solely neuropsychiatric symptoms. "This case emphasises that neurosyphilis still has to be considered in the differential diagnosis within the context of psychiatric ...
People are most likely to suffer their first depressive episode between the ages of 30 and 40, and there is a second, smaller peak of incidence between ages 50 and 60. [8] The risk of major depression is increased with neurological conditions such as stroke , Parkinson's disease , or multiple sclerosis and during the first year after childbirth ...
Endogenous depression is an atypical subclass of major depressive disorder (clinical depression). It could be caused by genetic and biological factors. [ 1 ] Endogenous depression occurs due to the presence of an internal (cognitive, biological) stressor instead of an external (social, environmental) stressor. [ 2 ]
In modern times, depression, more often severe cases, is more noted as an absence of pleasure, with feelings of emptiness and flatness. [4] In the United States and Canada, the costs associated with major depression are comparable to those related to heart disease, diabetes, and back problems and are greater than the costs of hypertension. [5]
For most, the feelings of sadness and emptiness pass with time. However, for some people, it can linger, intensify and even dominate their lives, crippling their ability to go through life.
[60] [61] As with the relationship between depression and inflammation as a whole, this relationship is thought to be bidirectional with both depression increasing one's risk of autoimmune disease and autoimmune disease increasing one's risk of depression. [60] However, it is also likely that this connection is also due in part to depression ...
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