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Metabolic alkalosis is an acid-base disorder in which the pH of tissue is elevated beyond the normal range (7.35–7.45). This is the result of decreased hydrogen ion concentration, leading to increased bicarbonate (HCO − 3), or alternatively a direct result of increased bicarbonate concentrations.
Alkalosis is the result of a process reducing hydrogen ion concentration of arterial blood plasma (alkalemia). In contrast to acidemia (serum pH 7.35 or lower), alkalemia occurs when the serum pH is higher than normal (7.45 or higher).
The skin of a cat deficient in zinc would likely have erythema and hair loss. The cat may have crusty, scaly skin on its limbs or tail. [1] The coat of the cat becomes dull. Similarly, copper can affect coat health of cats; deficiencies will cause fading of coat color and weakened skin, leading to lesions. [3] [4]
Feline cutaneous asthenia is a rare inheritable skin disease of cats characterised by abnormal elasticity, stretching, and improper healing of the skin. Pendulous wing-like folds of skin form on the cat's back, shoulders and haunches. Even stroking the cat can cause the skin to stretch and tear.
Diagnosis of contraction alkalosis is made by correlating laboratory data with clinical history and examination. Metabolic alkalosis in the presence of decreased effective circulatory volume, loop diuretic use, or other causes of intravascular depletion such as profound diarrhea should raise suspicion for contraction alkalosis as a likely etiology in the absence of other causes.
Cats can be affected by hyperaldosteronism. The most common signs in cats are muscle weakness and loss of eyesight, although only one of these signs may be present. [ 12 ] Muscle weakness is due to low potassium concentrations in the blood, and signs of muscle weakness, such as being unable to jump, may be intermittent. [ 12 ]
Lentigo in cats is a common dermatological condition characterized by the presence of small, flat, brownish spots on the skin — particularly around the lips, nose, and eyelid margins.
In summary, hyperaldosteronism causes hypernatremia, hypokalemia, and metabolic alkalosis. [ 13 ] Finer notes on aldosterone include the fact that it stimulates sodium-potassium ATPase in muscle cells , increasing intracellular potassium and also increases sodium reabsorption all along the intestine and nephron , possibly due to widespread ...