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Although the host may not be experiencing symptoms, the virus can still be passed on to others. It is also possible for the infection to become symptomatic after this incubation period. Whether the host is showing symptoms or not, opportunistic infections can take advantage of the weakened immune system and cause further complications. [20]
[26] [32] When either of these occur, memory T cells no longer recognize the virus, so people are not immune to the dominant circulating strain. [31] [32] For both influenza and norovirus, epidemics temporarily induce herd immunity until a new dominant strain emerges, causing successive waves of epidemics.
At least a third of the people who are infected with the virus do not develop noticeable symptoms at any point in time. [28] [29] [30] Asymptomatic carriers tend not to get tested. [30] [31] [32] Persons with asymptomatic COVID-19 infection can have the same viral load as symptomatic and presymptomatic cases, and are able to transmit the virus. [8]
There are tons of reasons why some people can't apologize. Still, ultimately, it doesn’t matter whether they’re afraid of looking weak or just really don’t want to take responsibility for ...
SARS-CoV-2 is the seventh known coronavirus to infect people, after 229E, NL63, OC43, HKU1, MERS-CoV, and the original SARS-CoV. [105] Like the SARS-related coronavirus implicated in the 2003 SARS outbreak, SARS‑CoV‑2 is a member of the subgenus Sarbecovirus (beta-CoV lineage B). [106] [107] Coronaviruses undergo frequent recombination. [108]
However, the absence of the symptom itself at an initial screening does not rule out COVID-19. Fever in the first week of a COVID-19 infection is part of the body's natural immune response; however in severe cases, if the infections develop into a cytokine storm the fever is counterproductive. As of September 2020, little research had focused ...
The original antigenic sin: When the body first encounters an infection it produces effective antibodies against its dominant antigens and thus eliminates the infection. But when it encounters the same infection, at a later evolved stage, with a new dominant antigen, with the original antigen now being recessive, the immune system will still produce the former antibodies against this old "now ...
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