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However, fluid overload can exacerbate acute kidney injury. The use of diuretics does not prevent or treat AKI even with the help of renal replacement therapy. The 2012 KDIGO (Kidney Disease: Improving Global Outcomes) guidelines stated that diuretics should not be used to treat AKI, except for the management of volume overload. [13]
The 2012 KDIGO (Kidney Disease: Improving Global Outcomes) guidelines stated that diuretics should not be used to treat acute kidney injury, except for the management of volume overload. Diuretics has not shown any benefits of preventing or treating acute kidney injury. [16]
Acute kidney injury was one of the most expensive conditions seen in U.S. hospitals in 2011, with an aggregated cost of nearly $4.7 billion for approximately 498,000 hospital stays. [48] This was a 346% increase in hospitalizations from 1997, when there were 98,000 acute kidney injury stays. [49]
"Summary of KDIGO 2012 CKD Guideline: behind the scenes, need for guidance, and a framework for moving forward". Kidney International. 85 (1): 49–61. doi: 10.1038/ki.2013.444. PMID 24284513. Co-author of textbook: “Chronic Kidney Disease: a practical guide to understanding and management”, ISBN 9780199549313
In nephrology, renal angina is a clinical methodology to risk stratify patients for the development of persistent and severe acute kidney injury (AKI). [1] The composite of risk factors and early signs of injury for AKI, renal angina is used as a clinical adjunct to help optimize the use of novel AKI biomarker testing.
Upon presentation of decreased renal function, it is recommended to perform a history and physical examination, as well as performing a renal ultrasound and a urinalysis. [ citation needed ] The most relevant items in the history are medications , edema , nocturia , gross hematuria , family history of kidney disease, diabetes and polyuria .
Kidney Disease Improving Global Outcomes – KDIGO 2012 Clinical Practice Guideline for the Evaluation and Management of Chronic Kidney Disease [58] A glomerular filtration rate (GFR) ≥ 60 mL/min/1.73 m 2 is considered normal without chronic kidney disease if there is no kidney damage present.
Acute tubular necrosis (ATN) is a medical condition involving the death of tubular epithelial cells that form the renal tubules of the kidneys.Because necrosis is often not present, the term acute tubular injury (ATI) is preferred by pathologists over the older name acute tubular necrosis (ATN). [1]