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The serotonin "chemical imbalance" theory of depression, proposed in the 1960s, [35] is not supported by the available scientific evidence. [ 35 ] [ 36 ] SSRIs alter the balance of serotonin inside and outside of neurons: their clinical antidepressant effect (which is robust in severe depression [ 37 ] ) is likely due to more complex changes in ...
This is an alphabetically sorted list of all mental disorders in the DSM-IV and DSM-IV-TR, along with their ICD-9-CM codes, where applicable. The DSM-IV-TR is a text revision of the DSM-IV. [1] While no new disorders were added in this version, 11 subtypes were added and 8 were removed. This list features both the added and removed subtypes.
There are several main groups. Antidepressants are used for the treatment of clinical depression as well as often for anxiety and other disorders. [30] Anxiolytics are used, generally short-term, for anxiety disorders and related problems such as physical symptoms and insomnia. [31]
weakness, or anxiety Large dose: loss of consciousness Perfluoroisobutene Gas: colorless Odor: none 1-4 hours Flu-like, also eye, nose, and throat irritation, and chest discomfort, or no symptoms Phosgene Gas: colorless Odor: decaying fruit, fresh-cut grass, 1-4 hours; small doses, 24-48 hours Burning eyes, sore throat, dry
This is a list of psychiatric medications used by psychiatrists and other physicians to treat mental illness or distress. The list is ordered alphabetically according to the condition or conditions, then by the generic name of each medication. The list is not exhaustive and not all drugs are used regularly in all countries.
One such cause is chemical imbalances in the brain, which can lead to irrational decisions and emotional pain. [8] For example, when the brain lacks serotonin, a chemical that regulates the brain's functioning, it can lead to depression, appetite changes, aggression, and anxiety. Another cause of mental distress can be exposure to severely ...
The pharmacology of antidepressants is not entirely clear.. The earliest and probably most widely accepted scientific theory of antidepressant action is the monoamine hypothesis (which can be traced back to the 1950s), which states that depression is due to an imbalance (most often a deficiency) of the monoamine neurotransmitters (namely serotonin, norepinephrine and dopamine). [1]
Many psychologists criticise psychiatry for focusing its attention primarily on symptoms and for assuming that relieving symptoms with drugs cures the problem. Unfortunately, in many cases when the drug treatment is ceased the symptoms recur. This suggests that drugs are not addressing the true cause of the problem. [4]