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  2. Edith Prentiss - Wikipedia

    en.wikipedia.org/wiki/Edith_Prentiss

    Prentiss had asthma, and in later life became diabetic. She started using a wheelchair in her late 40s as her asthma became severe. [1] Awards and honors

  3. Asthma - Wikipedia

    en.wikipedia.org/wiki/Asthma

    Non-atopic asthma, also known as intrinsic or non-allergic, makes up between 10 and 33% of cases. There is negative skin test to common inhalant allergens. Often it starts later in life, and women are more commonly affected than men. Usual treatments may not work as well. [129]

  4. Ann Woolcock - Wikipedia

    en.wikipedia.org/wiki/Ann_Woolcock

    Ann Janet Woolcock (11 December 1937 – 17 February 2001) was an Australian respiratory physician–scientist and one of the world's leading asthma experts. She contributed greatly to the field of asthma research and founded the Institute of Respiratory Medicine, Sydney, which is now known as the Woolcock Institute of Medical Research.

  5. Joyce Vincent - Wikipedia

    en.wikipedia.org/wiki/Joyce_Vincent

    She died sometime in December 2003. Her remains were discovered on 25 January 2006, with the cause of death believed to be either an asthma attack or complications from a recent peptic ulcer. Vincent's life and death were the topic of Dreams of a Life, a 2011 docudrama film. The film and Vincent's life inspired musician Steven Wilson's album ...

  6. Asthma-related microbes - Wikipedia

    en.wikipedia.org/wiki/Asthma-related_microbes

    Furthermore, children who experience severe viral respiratory infections early in life have a high possibility of having asthma later in their childhood. These viral respiratory infections are mostly caused by respiratory syncytial virus (RSV) and human rhinovirus (HRV). Although RSV infections increase the risk of asthma in early childhood ...

  7. Airway remodelling - Wikipedia

    en.wikipedia.org/wiki/Airway_remodelling

    Airway remodelling is a multifaceted process involving multiple airway tissues. These include goblet cell hyperplasia, leading to increased mucus production, and airway smooth muscle hypertrophy (or smooth muscle cell hyperplasia), leading to the release of pro-inflammatory and pro-fibrotic messengers contributing to subepithelial fibrosis.

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