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Lance–Adams syndrome (LAS) is a sequela of hypoxic encephalopathy due to respiratory arrest, airway obstruction, cardiac arrest, etc., several days after the onset of hypoxic encephalopathy. A condition that presents with functional myoclonus associated with increased cortical excitability in a few weeks.
Action myoclonus is the most disabling form of myoclonus and can affect the arms, legs, face, and even the voice. It is often associated with tonic-clonic seizures and diffuse neuronal disease such as post-hypoxic encephalopathy , uremia , and the various forms of PME, although, in the case of focal cerebral damage, the disease may be ...
Together with the Australian neurologist James Waldo Lance he described posthypoxic myoclonus, later called Lance-Adams syndrome. [11] Adams, in collaboration with Canadian neurologist Dr. C. Miller Fisher, made contributions to the field of cerebrovascular disease, the syndrome of "transient global amnesia" in 1964, [12] and in 1965 he ...
Symptoms often include action or stimuli induced myoclonus, seizures, neuropathy, cognitive decline, and spike and wave or no cerebral discharges. [4] The prognosis of those diagnosed with PME is poor. The person often becomes reliant on a wheelchair, enters a vegetative state due to myoclonus, and has a shortened life expectancy. [4] [3]
Familial adult myoclonus Epilepsy (FAME) This is a condition characterized by the repetition of non-coding sequences and has been identified using various abbreviations. Initially, it was associated with four primary gene locations: FAME1 (8q23.3–q24.1), FAME2 (2p11.1–q12.1), FAME3 (5p15.31–p15.1), and FAME4 (3q26.32–3q28).
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James Waldo Lance AO, CBE (1926–2019), often referred to as James Lance and James W. Lance, was an Australian neurologist.He was the founder of the School of Neurology at the University of New South Wales and president of the International Headache Society in 1987–89, [1] and a "world authority on the diagnosis and treatment" of headache and migraine.