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Erythema annulare centrifugum (EAC), is a descriptive term for a class of skin lesion [2] presenting redness in a ring form (anulare) that spreads from a center (centrifugum). It was first described by Darier in 1916.
This type of rash can be as a result of large doses of niacin or no-flush niacin (2000 – 2500 mg), [citation needed] used for the management of low HDL cholesterol. [4] This type of rash can also be a symptom of Sea bather's eruption. This stinging, pruritic, maculopapular rash affects swimmers in some Atlantic locales (e.g., Florida ...
Gottron's sign is a pathognomonic cutaneous manifestation associated with dermatomyositis (DM), which is an inflammatory disorder affecting the skin and muscles. [1] The primary lesion of dermatomyositis appears as a violaceous, macular erythema with a symmetric distribution, which may progress and become poikilodermatous (atrophic with telangiectasia and pigmentary changes) and indurated (as ...
[1] [2] The rash consists of macular lesions that are red and usually 2–10 mm in diameter but may be confluent in places. [3] A morbilliform rash is a rose-red flat or slightly elevated (maculopapular) eruption, showing circular or elliptical lesions varying in diameter from 1 to 3 mm, with healthy-looking skin intervening. [citation needed]
Confluent erythematous and edematous patches on cheeks ("slapped cheek") for 1–4 days followed by a "lacy," reticular, erythematous rash on the body. Roseola infantum (exanthem subitum) "sixth disease" HHV-6 and HHV-7: Rapid onset of erythematous, blanching macules and papules surrounded by white halos on the trunk after 3–5 days of high fever.
Other symptoms to note: Drug rashes can be a side effect of or a reaction to a new medication; almost any medication can cause a drug rash, but antibiotics and NSAIDs are the most common culprits ...
The presence of systemic involvement or clinical symptoms determines how individuals with TMEP are treated. There is no gold standard medication for the treatment of TMEP, thus it is critical to identify and avoid conditions that trigger mast cell destruction, such as sunshine, severe temperatures, alcohol , and narcotics .
Diagnosis is typically based on some combination of symptoms, blood tests, electromyography, and muscle biopsies. [2] Eighty percent of adults [5] and sixty percent of children with juvenile dermatomyositis have a myositis-specific antibody (MSA). [6] Although no cure for the condition is known, treatments generally improve symptoms. [1]