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The book is a collection of stories of doctors and patients showing that the human brain is capable of undergoing change, including stories of recovering use of paralyzed body parts, deaf people learning to hear, and others getting relief from pain using exercises to retrain neural pathways.
Doidge has written over 170 articles, a combination of academic, scientific and popular pieces. He has been sole author of academic papers on neuroplasticity, human limitations and notions of perfectibility, psychotherapy treatment outcomes, dreams about animals, Schizoid personality disorder and trauma, [8] psychoanalysis, and neuroscience, such as a popular article he wrote in 2006 for ...
Neuroplasticity, also known as neural plasticity or just plasticity, is the ability of neural networks in the brain to change through growth and reorganization. Neuroplasticity refers to the brain's ability to reorganize and rewire its neural connections, enabling it to adapt and function in ways that differ from its prior state.
How the brain changes. Brain plasticity science is the study of a physical process. Gray matter can actually shrink or thicken; neural connections can be forged and refined or weakened and severed.
In neuroscience, homeostatic plasticity refers to the capacity of neurons to regulate their own excitability relative to network activity. The term homeostatic plasticity derives from two opposing concepts: 'homeostatic' (a product of the Greek words for 'same' and 'state' or 'condition') and plasticity (or 'change'), thus homeostatic plasticity means "staying the same through change".
Jeffrey Schwartz and Beverly Beyette, Brain Lock: Free Yourself from Obsessive-Compulsive Behavior, New York: Regan Books, 1997. ISBN 0-06-098711-1. [9] Jeffrey Schwartz and Sharon Begley, The Mind and the Brain: Neuroplasticity and the power of mental force, New York: Regan Books, 2002. ISBN 0-06-039355-6.
Two molecular mechanisms for synaptic plasticity involve the NMDA and AMPA glutamate receptors. Opening of NMDA channels (which relates to the level of cellular depolarization) leads to a rise in post-synaptic Ca 2+ concentration and this has been linked to long-term potentiation, LTP (as well as to protein kinase activation); strong depolarization of the post-synaptic cell completely ...
In 1973, M. M. Taylor [1] suggested that if synapses were strengthened for which a presynaptic spike occurred just before a postsynaptic spike more often than the reverse (Hebbian learning), while with the opposite timing or in the absence of a closely timed presynaptic spike, synapses were weakened (anti-Hebbian learning), the result would be an informationally efficient recoding of input ...