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The term trophic is from Ancient Greekτροφικός (trophikós) meaning "pertaining to food or nourishment", here used to mean "growth"; this is the same origin as atrophy. This should not be confused with tropic, as in the similar-sounding tropic hormone – the words and concepts are both unrelated.
The term tropic is from Ancient Greek τροπικός (tropikós), in the sense "of or pertaining to a turn or change", meaning "causing a change, affecting"; this is the same origin as tropic and trope. This should not be confused with trophic, as in similar-sounding trophic hormone – the words and concepts are both unrelated. [3]
The hypothalamus produces the hormones oxytocin and vasopressin in its endocrine cells (left). These are released at nerve endings in the posterior pituitary gland and then secreted into the systemic circulation. The hypothalamus releases tropic hormones into the hypophyseal portal system to the anterior pituitary (right).
The main release-inhibiting hormones or inhibiting hormones are as follows: The hypothalamus uses somatostatin to tell the pituitary to inhibit somatotropin and to tell the gastrointestinal tract to inhibit various gastrointestinal hormones. There are various other inhibiting factors that also have tropic endocrine inhibition activity.
Non-tropic hormones are thus often the last piece of a larger process and chain of hormone secretion. Both tropic and non-tropic hormones are necessary for proper endocrine function. For example, if ACTH (Adrenocorticotropin hormone; a tropic hormone) is inhibited, cortisol can no longer be released because the chain reaction has been ...
For Hadley, the key takeaway is that making nutrition changes for the sake of your hormones should only be done based on your body’s unique needs. “While there are foundational strategies that ...
The migration of the growth cone is mediated through the interaction of numerous trophic and tropic factors; netrins, slits, ephrins and semaphorins are four well-studied tropic cues . The growth cone is capable of modifying its sensitivity to these guidance molecules as it migrates to its target; this sensitivity regulation is an important ...
Upon ligand binding, the receptor undergoes conformation changes that stimulate the enzyme adenylyl cyclase, which leads to an increase in intracellular cAMP [11] and subsequent activation of protein kinase A. ACTH influences steroid hormone secretion by both rapid short-term mechanisms that take place within minutes and slower long-term actions.