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The name "pyogenic granuloma" is misleading, as it is neither pyogenic or a true granuloma. Rather, it is a capillary hemangioma of lobular subtype, which is why such a lesion is prone to bleeding. [5] It is also not truly pyogenic (pus-producing), as the cause is hormonal or traumatic and has no association with infection or pus production.
This type of epulis is neither pyogenic ("pus producing") nor a true granuloma, but it is a vascular lesion.About 75% of all pyogenic granulomas occur on the gingiva, [2] growing beneath the gingival margin, [8] although they may also occur elsewhere in the mouth or other parts of the body (where the term epulis is inappropriate).
Peripheral giant-cell granuloma (PGCG) is an oral pathologic condition that appears in the mouth as an overgrowth of tissue due to irritation or trauma. Because of its overwhelming incidence on the gingiva , the condition is associated with two other diseases , pyogenic granuloma and peripheral ossifying fibroma .
Instead, the three are associated with each other because they appear frequently on gingiva: pyogenic granuloma and peripheral giant cell granuloma. Some researchers believe peripheral ossifying fibromas to be related to pyogenic fibromas and, in some instances, are the result of a pyogenic granuloma which has undergone fibrosis and calcification.
English: Histopathology of pyogenic granuloma - high magnification. HE stain. Annotated are major features: Endothelial cell clusters, inflammation, variable amount of mitoses, and edema. - Christopher S. Hale, M.D.. Skin nonmelanocytic tumor - Vascular tumors - Capillary / pyogenic granuloma. PathologyOultines. Topic Completed: 1 August 2012.
Foreign body granuloma formation consists of protein adsorption, macrophages, multinucleated foreign body giant cells (macrophage fusion), fibroblasts, and angiogenesis. It has also been proposed that the mechanical property of the interface between an implant and its surrounding tissues is critical for the host response.
Gingival enlargement has a multitude of causes. The most common is chronic inflammatory gingival enlargement, when the gingivae are soft and discolored. This is caused by tissue edema and infective cellular infiltration caused by prolonged exposure to bacterial plaque, and is treated with conventional periodontal treatment, such as scaling and root planing.
Necrotizing periodontal diseases are non-contagious infections but may occasionally occur in epidemic-like patterns due to shared risk factors. The milder form, necrotizing ulcerative gingivitis (also termed "trench mouth"), [9] is characterized by painful, bleeding gums and ulceration and necrosis of the interdental papilla.