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It is now thought that this white lesion on the tongue represented syphilitic glossitis, [30] a condition not included in the modern definitions of oral leukoplakia. Since then, the word leukoplakia has been incorporated into the names for several other oral lesions (e.g. candidal leukoplakia , now more usually termed hyperplastic candidiasis ...
The lesion is a white patch, which almost exclusively occurs on the lateral surfaces of the tongue, although rarely it may occur on the buccal mucosa, soft palate, pharynx or esophagus. [5] The lesion may grow to involve the dorsal surface of the tongue. The texture is vertically corrugated ("hairy") or thickly furrowed and shaggy in appearance.
Transient lingual papillitis (TLP) is a medical term for painful, hypertrophic, red, and white lingual papillae on the tongue. [3] TLP is also called lie bumps and fungiform papillary glossitis. This condition has four types: classic form, transient u-shaped lingual papillitis, papulokeratotic variant, and eruptive lingual papillitis. [ 4 ]
This is an elliptical or rhomboid lesion in the center of the dorsal tongue, just anterior (in front) of the circumvallate papillae. The area is depapillated, reddened (or red and white) and rarely painful. There is frequently Candida species in the lesion, sometimes mixed with bacteria. [13]
Leukoedema lesions disappear when the mucosa is stretched, which helps to differentiate it from other white lesions in the mouth. [2] The differential diagnosis is with leukoplakia , oral candidiasis , oral lichen planus , white sponge nevus , morsicatio buccarum , [ 3 ] hereditary benign intraepithelial dyskeratosis and dyskeratosis congenita.
Tongue lesions are very common. For example, in the United States one estimated point prevalence was 15.5% in adults. [10] Tongue lesions are more common in persons who wear dentures and tobacco users. [10] The most common tongue conditions are geographic tongue, followed by fissured tongue and hairy tongue. [10]
White sponge nevus (WSN) is an extremely rare autosomal dominant [4] condition of the oral mucosa (the mucous membrane lining of the mouth). It is caused by one or more mutations in genes coding for keratin , which causes a defect in the normal process of keratinization of the mucosa.
The lesions are harmless; no treatment is indicated beyond reassurance unless the person requests it. The most common and simple treatment is the construction of a specially made acrylic prosthesis that covers the biting surfaces of the teeth and protects the cheek, tongue, and labial mucosa (an occlusal splint). This is either employed in the ...
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