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Repolarization of the ventricle happens in the opposite direction of depolarization and is negative current, signifying the relaxation of the cardiac muscle of the ventricles. But this negative flow causes a positive T wave; although the cell becomes more negatively charged, the net effect is in the positive direction, and the ECG reports this ...
Ventricular relaxation, or diastole, follows repolarization of the ventricles and is represented by the T wave of the ECG. It too is divided into two distinct phases and lasts approximately 430 ms. [1] During the early phase of ventricular diastole, as the ventricular muscle relaxes, pressure on the remaining blood within the ventricle begins ...
The P wave is a summation wave generated by the depolarization front as it transits the atria. Normally the right atrium depolarizes slightly earlier than left atrium since the depolarization wave originates in the sinoatrial node, in the high right atrium and then travels to and through the left atrium.
Repolarization of the atria occurs at the same time as the generation of the QRS complex, but it is not detected by the ECG since the tissue mass of the ventricles is so much larger than that of the atria. Ventricular contraction occurs between ventricular depolarization and repolarization.
Schematic representation of normal ECG. In electrocardiography, the ST segment connects the QRS complex and the T wave and has a duration of 0.005 to 0.150 sec (5 to 150 ms). ...
Cardiac (ventricular) systole: Both AV valves (tricuspid in the right heart (light-blue), mitral in the left heart (pink)) are closed by back-pressure as the ventricles are contracted and their blood volumes are ejected through the newly-opened pulmonary valve (dark-blue arrow) and aortic valve (dark-red arrow) into the pulmonary trunk and ...
In non-pacemaker cells (i.e. ventricular cells), this is produced predominantly by the activation of Na + channels, which increases the membrane conductance (flow) of Na + (g Na). These channels are activated when an action potential arrives from a neighbouring cell, through gap junctions. When this happens, the voltage within the cell ...
The net result is that, while contraction causes ventricular pressures to rise sharply, there is no overall change in volume because of the closed valves. The isovolumetric contraction phase lasts about 0.05 seconds, [ 1 ] but this short period of time is enough to build up a sufficiently high pressure that eventually overcomes that of the ...
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