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The current formal name established in the third edition of the International Classification of Sleep Disorders (ICSD-3) is delayed sleep-wake phase disorder. Earlier, and still common, names include delayed sleep phase disorder (DSPD), delayed sleep phase syndrome (DSPS), delayed sleep phase type (DSPT), and circadian rhythm sleep disorder. [37]
Sleep inertia is a physiological state of impaired cognitive and sensory-motor performance that is present immediately after awakening. It persists during the transition of sleep to wakefulness, where an individual will experience feelings of drowsiness, disorientation and a decline in motor dexterity.
Sleep paralysis is a state, during waking up or falling asleep, in which a person is conscious but in a complete state of full-body paralysis. [1] [2] During an episode, the person may hallucinate (hear, feel, or see things that are not there), which often results in fear. [1] [3] Episodes generally last no more than a few minutes. [2]
1. Fatigue. Research indicates that daytime sleepiness is the most obvious and common sign of sleep debt.If you feel groggy even after you’ve been awake for a while or if you find yourself ...
Other risk factors for sleep eating include a family history of eating disorders of any kind as well as the person’s sex, Schenck said. “Sleep-related eating is like 70% female predominant ...
EDS can be a symptom of a number of factors and disorders. Specialists in sleep medicine are trained to diagnose them. Some are: Insufficient quality or quantity of night time sleep [5] Obstructive sleep apnea [6] Misalignments of the body's circadian pacemaker with the environment (e.g., jet lag, shift work, or other circadian rhythm sleep ...
“Encourage activities and exposure to natural light during the day to support healthy sleep-wake cycles.” It's also crucial to try to avoid overstimulation in the evenings by limiting loud ...
Fatal insomnia is an extremely rare neurodegenerative prion disease that results in trouble sleeping as its hallmark symptom. [2] The majority of cases are familial (fatal familial insomnia [FFI]), stemming from a mutation in the PRNP gene, with the remainder of cases occurring sporadically (sporadic fatal insomnia [sFI]).