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Although ARDS can present with pulmonary edema (fluid accumulation), it is a distinct clinical syndrome that is not synonymous with pulmonary edema. Direct lung injury Acute lung injury may cause pulmonary edema directly through injury to the vasculature and parenchyma of the lung, causes include:
The pathophysiology of acute respiratory distress syndrome involves fluid accumulation in the lungs not explained by heart failure (noncardiogenic pulmonary edema). It is typically provoked by an acute injury to the lungs that results in flooding of the lungs' microscopic air sacs responsible for the exchange of gases such as oxygen and carbon dioxide with capillaries in the lungs. [1]
A pleural effusion is accumulation of excessive fluid in the pleural space, the potential space that surrounds each lung.Under normal conditions, pleural fluid is secreted by the parietal pleural capillaries at a rate of 0.6 millilitre per kilogram weight per hour, and is cleared by lymphatic absorption leaving behind only 5–15 millilitres of fluid, which helps to maintain a functional ...
In 1994, a new definition was recommended by the American-European Consensus Conference Committee [6] [10] which recognized the variability in severity of pulmonary injury. [51] The definition required the following criteria to be met: acute onset, persistent dyspnea; bilateral infiltrates on chest radiograph consistent with pulmonary edema
Other causes of pulmonary edema that require rapid intervention and should be considered first include fluid overload, brain injury, and anaphylaxis. If when considering these differentials, there is no evidence for administration of excessive fluids, no focal signs suggesting a brain injury, and so signs of allergic reaction, one can then ...
The edema contributes to the deposition of a hyaline membrane (composed of dead cells, surfactant, and proteins) along the alveolar walls. Hyaline membranes are characteristic of DAD. The edema interferes with the naturally occurring surfactant, which is critical for reducing surface tension and allowing alveoli to remain open and allow air in ...
An increase in Pi causes extraalveolar blood vessels to reduce in caliber, in turn causing blood flow to decrease (extraalveolar blood vessels are those blood vessels outside alveoli). Intraalveolar blood vessels (pulmonary capillaries) are thin walled vessels adjacent to alveoli which are subject to the pressure changes described by zones 1-3.
Causes of Kerley B lines include pulmonary edema, lymphangitis carcinomatosa and malignant lymphoma, viral and mycoplasmal pneumonia, interstitial pulmonary fibrosis, pneumoconiosis, and sarcoidosis. They can be an evanescent sign on the chest x-ray of a patient in and out of heart failure.