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Ciguatoxin is produced by Gambierdiscus toxicus, a type of dinoflagellate. The phenomenon occurs in the Caribbean Sea, Hawaii, and coastal Central America. The toxin usually accumulates in the skin, head, viscera, and roe of big reef fish like grouper, wrasse, triggerfish, lionfish, and amberjack.
Signal transmission from nerve to muscle at the motor end plate. The neuromuscular junction is the synapse that is formed between an alpha motor neuron (α-MN) and the skeletal muscle fiber. In order for a muscle to contract, an action potential is first propagated down a nerve until it reaches the axon terminal of the motor neuron.
Depolarization is essential to the function of many cells, communication between cells, and the overall physiology of an organism. Action potential in a neuron, showing depolarization, in which the cell's internal charge becomes less negative (more positive), and repolarization, where the internal charge returns to a more negative value.
Ciguatoxin 1 or CTX-1 is a toxic chemical compound, the most common and potent type in the group of ciguatoxins. It is a large molecule consisting of polycyclic polyethers that can be found in certain types of fish in the Pacific Ocean. The compound is produced by Dinoflagellates Gambierdiscus toxicus and is passed on through the food chain by ...
Ciguatoxin has no taste or smell, and cannot be destroyed by conventional cooking. [2] There is no specific treatment for ciguatera fish poisoning once it occurs. [2] Mannitol may be considered, but the evidence supporting its use is not very strong. [1] Gabapentin or amitriptyline may be used to treat some of the symptoms. [2]
The action potential in a normal skeletal muscle cell is similar to the action potential in neurons. [61] Action potentials result from the depolarization of the cell membrane (the sarcolemma), which opens voltage-sensitive sodium channels; these become inactivated and the membrane is repolarized through the outward current of potassium ions ...
In addition, Toulouse seemed to have a lively nightlife, which makes sense considering the many nearby universities. It was a bit too much for me, especially since this meant we heard many loud ...
Glutamate released from the upper motor neurons triggers depolarization in the lower motor neurons in the anterior grey column, which in turn causes an action potential to propagate the length of the axon to the neuromuscular junction where acetylcholine is released to carry the signal across the synaptic cleft to the postsynaptic receptors of the muscle cell membrane, signaling the muscle to ...