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The Autism – Tics, ADHD, and other Comorbidities Inventory (A–TAC) is a psychological measure used to screen for other conditions occurring with tics. [ 1 ] [ 2 ] Along with tic disorders , it screens for autism spectrum disorders, attention deficit hyperactivity disorder (ADHD) and other conditions with onset in childhood. [ 1 ]
A symptom of some autistic children is the struggle to produce spontaneous speech. Studies have shown that in some cases echolalia is used as a coping mechanism allowing an autistic person to contribute to a conversation when unable to produce spontaneous speech. [ 2 ]
Autistic children are less likely to make requests or share experiences and more likely to simply repeat others' words . [106] The CDC estimated in 2015 that around 40% of autistic children do not speak at all. [107] Autistic adults' verbal communication skills largely depend on when and how well speech is acquired during childhood. [103]
Although tic disorders are commonly considered to be childhood syndromes, tics occasionally develop during adulthood; adult-onset tics often have a secondary cause. [33] Tics that begin after the age of 18 are not diagnosed as Tourette's syndrome, but may be diagnosed as an "other specified" or "unspecified" tic disorder. [24]
Most autistic children displayed moderately less attachment security than neurotypical children, although this difference disappears in children with higher mental development or less pronounced autistic traits. [24] Children with high-functioning autism have more intense and frequent loneliness compared to non-autistic peers, despite the ...
Palilalia must be differentiated from other complex tic disorders (such as echolalia), stuttering, [10] and logoclonia. In contrast to stuttering or logoclonia, palilalic repetitions tend to consist of complete sections of words or phrases, [ 5 ] are often repeated many times, [ 11 ] and the speaker has no difficulty initiating speech.
Echopraxia is a typical symptom of Tourette syndrome but causes are not well elucidated. [1]Frontal lobe animation. One theoretical cause subject to ongoing debate surrounds the role of the mirror neuron system (MNS), a group of neurons in the inferior frontal gyrus (F5 region) of the brain that may influence imitative behaviors, [1] but no widely accepted neural or computational models have ...
Theories about the cause of self-injurious behavior in children with developmental delay, including autistic children, include: [91] Frequency or continuation of self-injurious behavior can be influenced by environmental factors (e.g., reward in return for halting self-injurious behavior). This theory does not apply to younger children with autism.