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Maltitol is a sugar alcohol (a polyol) used as a sugar substitute and laxative. It has 75–90% of the sweetness of sucrose (table sugar) and nearly identical properties, except for browning . It is used to replace table sugar because it is half as calorific , does not promote tooth decay, and has a somewhat lesser effect on blood glucose .
Similarly, due to a 1985 report by the E.U. Scientific Committee on Food which states that "ingesting 50 g a day of xylitol can cause diarrhea", tabletop sweeteners (as well as other products containing xylitol) are required to display the warning "Excessive consumption may induce laxative effects".
Bile acid malabsorption (BAM), known also as bile acid diarrhea, is a cause of several gut-related problems, the main one being chronic diarrhea.It has also been called bile acid-induced diarrhea, cholerheic or choleretic enteropathy, bile salt diarrhea or bile salt malabsorption.
Causes of Type 1 Diabetes. ... Aside from knowing the signs of diabetes in women or the most common diabetes symptoms men get, you may be curious if there are ways to prevent getting diabetes in ...
Lyon says this can be beneficial for people living with type 2 diabetes or metabolic issues like obesity. But the World Health Organization warns against using non-sugar sweeteners for weight ...
Erythritol is a sugar alcohol. It is 60–70% as sweet as sugar and almost noncaloric. Sugar alcohols (also called polyhydric alcohols, polyalcohols, alditols or glycitols) are organic compounds, typically derived from sugars, containing one hydroxyl group (−OH) attached to each carbon atom.
The symptoms of an episode of diabetic ketoacidosis usually evolve over a period of about 24 hours. Predominant symptoms are nausea and vomiting, pronounced thirst, excessive urine production and abdominal pain that may be severe. [13] [14] In severe DKA, breathing becomes rapid and of a deep, gasping character, called "Kussmaul breathing".
Some FODMAPs, such as fructose, are readily absorbed in the small intestine of humans via GLUT receptors. [19] Absorption thus depends on the appropriate expression and delivery of these receptors in the intestinal enterocyte to both the apical surface, contacting the lumen of the intestine (e.g., GLUT5), and to the basal membrane, contacting the blood (e.g., GLUT2). [19]