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1–24 hours – the injury is moderate in severity and full recovery is expected. The patient may experience some minor post-concussive symptoms (e.g. headaches, dizziness). 1–7 days – the injury is severe, and recovery may take weeks to months. The patient may be able to return to work, but may be less capable than before the injury.
Hyperthymesia, also known as hyperthymestic syndrome or highly superior autobiographical memory (HSAM), is a condition that leads people to be able to remember an abnormally large number of their life experiences in vivid detail.
Drug-induced amnesia is amnesia caused by drugs. Amnesia may be therapeutic for medical treatment or for medical procedures, or it may be a side-effect of a drug, such as alcohol, or certain medications for psychiatric disorders, such as benzodiazepines. [1] It is seen also with slow acting parenteral general anaesthetics. [citation needed]
Motivated forgetting is a theorized psychological behavior in which people may forget unwanted memories, either consciously or unconsciously. [1] It is an example of a defence mechanism, since these are unconscious or conscious coping techniques used to reduce anxiety arising from unacceptable or potentially harmful impulses thus it can be a defence mechanism in some ways. [2]
An example of a subgroup of people who are often exposed to TBI are individuals who are involved in high-contact sports. Research on football players takes a closer look at some of the implications to their high-contact activities. Enduring consistent head injuries can have an effect on the neural consolidation of memory. [17]
Drug-induced amnesia is the idea of selectively losing or inhibiting the creation of memories using drugs. Amnesia can be used as a treatment for patients who have experienced psychological trauma or for medical procedures where full anesthesia is not an option. Drug-induced amnesia is also a side-effect of other drugs like alcohol and rohypnol.
Damage to the frontal or anterior temporal regions have been described to be associated with disproportionate RA. Studies have illustrated that during PTA, head injury patients showed accelerated forgetting of learned information. On the other hand, after PTA, forgetting rates were normal. [8]
Due to a lack of evidence for the concept of repressed and recovered memories, mainstream clinical psychologists have stopped using these terms. Clinical psychologist Richard McNally stated: "The notion that traumatic events can be repressed and later recovered is the most pernicious bit of folklore ever to infect psychology and psychiatry.