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  2. Fibrinogen - Wikipedia

    en.wikipedia.org/wiki/Fibrinogen

    Fibrinogen is made and secreted into the blood primarily by liver hepatocyte cells. Endothelium cells are also reported to make small amounts of fibrinogen, but this fibrinogen has not been fully characterized; blood platelets and their precursors, bone marrow megakaryocytes, while once thought to make fibrinogen, are now known to take up and store but not make the glycoprotein.

  3. Hyperfibrinolysis - Wikipedia

    en.wikipedia.org/wiki/Hyperfibrinolysis

    Moreover, hyperfibrinolysis may be associated with blood brain barrier impairment, a plasmin-dependent effect due to an increased generation of bradykinin. [8] Bleeding is caused by the generation of fibrinogen degradation products which interfere with regular fibrin polymerization and inhibit platelet aggregation. Moreover, plasmin which is ...

  4. Dysfibrinogenemia - Wikipedia

    en.wikipedia.org/wiki/Dysfibrinogenemia

    Fibrinogen is a glycoprotein made and secreted into the blood primarily by liver hepatocyte cells. Endothelium cells also make what appears to be small amounts of fibrinogen but this fibrinogen has not been fully characterized; blood platelets and their precursors, bone marrow megakaryocytes, although once thought to make fibrinogen, are now known to take up and store but not make the ...

  5. Fibrin monomer - Wikipedia

    en.wikipedia.org/wiki/Fibrin_monomer

    Levels of fibrin monomers can be measured using blood tests and can serve as a marker of in vivo fibrinogenesis and coagulation activation. [ 1 ] [ 2 ] [ 3 ] They may be useful in the evaluation of hypercoagulability , [ 1 ] as reflected in research studies done using fibrin monomers.

  6. Coagulation activation marker - Wikipedia

    en.wikipedia.org/wiki/Coagulation_activation_marker

    Coagulation activation markers are biomarkers of net activation of coagulation and fibrinolysis. [1] [2] Examples include prothrombin fragment 1+2 (F1+2), thrombin–antithrombin complex (TAT), fibrinopeptide A (FpA), fibrin monomers (FMs), plasmin-α 2-antiplasmin complex (PAP), activated protein C–protein C inhibitor (APC-PCI), and D-dimer (DD).

  7. List of fibrinogen disorders - Wikipedia

    en.wikipedia.org/wiki/List_of_fibrinogen_disorders

    The incorrectly glycosalated fibrinogen is dysfunctional and may cause pathological episodes of bleeding and/or blood clotting. [5] Congenital hypodysfibrinogenemia, an inherited disorder in which low levels of fibrinogen composed at least in part of a dysfunctional fibrinogen may cause pathological episodes of bleeding or blood clotting. [6]

  8. Simple blood test could predict a person’s heart disease risk ...

    www.aol.com/news/simple-blood-test-could-predict...

    All of the women had blood tests done at the beginning of the study to measure their LDL cholesterol, Lp(a) and C-reactive protein levels, a marker of inflammation in the body.

  9. D-dimer - Wikipedia

    en.wikipedia.org/wiki/D-dimer

    D-dimer formation. Shown are fibrinogen, with its one E domain and two D domains, acted upon in cascade, by the following enzymes: Thrombin, to create a mesh of fibrin protofibrils; Factor XIII to crosslink the fibrin mesh (linking protofibril D domains), the scaffold for clot formation; Plasmin, whose action in fibrinolysis produces fibrin degradation products (FDPs), the smallest of which ...