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The mylohyoid nerve needs to be blocked during local anaesthesia of the mandibular (lower) teeth to prevent pain during oral procedures. [ 5 ] [ 6 ] It may not be anaesthetised during a block of the inferior alveolar nerve , causing pain.
The inferior alveolar nerves supply sensation to the lower teeth, [2]: 519 and, via the mental nerve, sensation to the chin and lower lip. [citation needed] The mylohyoid nerve is a motor nerve supplying the mylohyoid and the anterior belly of the digastric. [citation needed] [contradictory]
Accessory mylohyoid muscles have been seen in some people, which have the same attachments, nerve supply, and function. [6] The mylohyoid muscle may also be split into an anterior portion and a posterior portion, with the sublingual gland occupying the space between these portions. [7] An area of herniation of the sublingual gland, blood ...
This is a list of major and frequently observed neurological disorders (e.g., Alzheimer's disease), symptoms (e.g., back pain), signs (e.g., aphasia) and syndromes (e.g., Aicardi syndrome). There is disagreement over the definitions and criteria used to delineate various disorders and whether some of these conditions should be classified as ...
Machado–Joseph disease (MJD), also known as Machado–Joseph Azorean disease, Machado's disease, Joseph's disease or spinocerebellar ataxia type 3 (SCA3), is a rare autosomal dominantly inherited neurodegenerative disease that causes progressive cerebellar ataxia, [1] [2] which results in a lack of muscle control and coordination of the upper and lower extremities. [3]
The margin of this opening is irregular; it presents in front a prominent ridge, surmounted by a sharp spine, the lingula of the mandible, which gives attachment to the sphenomandibular ligament; at its lower and back part is a notch from which the mylohyoid groove runs obliquely downward and forward, and lodges the mylohyoid vessels and nerve. [6]
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Hirayama disease, also known as monomelic amyotrophy (MMA), [1] [2] is a rare motor neuron disease first described in 1959 in Japan. Its symptoms usually appear about two years after adolescent growth spurt and is significantly more common in males, with an average age of onset between 15 and 25 years.