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Like many other medical conditions, obesity is the result of an interplay between environmental and genetic factors. [2] [3] Studies have identified variants in several genes that may contribute to weight gain and body fat distribution, although only in a few cases are genes the primary cause of obesity.
The commission said 18 factors can support a diagnosis of obesity. Examples include breathlessness, high blood pressure, knee or hip pain, reduced range of motion due to excess body fat, heart ...
Certain genetic conditions can also cause obesity. For example, Prader-Willi syndrome is an inherited disorder that leads to, among other complications, intellectual disability, a short stature ...
Fat mass and obesity-associated protein, also known as alpha-ketoglutarate-dependent dioxygenase FTO, is an enzyme that in humans is encoded by the FTO gene located on chromosome 16. As one homolog in the AlkB family proteins, it is the first messenger RNA (mRNA) demethylase that has been identified. [ 5 ]
16p11.2 deletion syndrome is a rare genetic condition caused by microdeletion on the short arm of chromosome 16. Most affected individuals experience global developmental delay and intellectual disability, as well as childhood-onset obesity. [1] 16p11.2 deletion is estimated to account for approximately 1% of autism spectrum disorder cases. [3] [4]
The following is a list of genetic disorders and if known, type of mutation and for the chromosome involved. Although the parlance "disease-causing gene" is common, it is the occurrence of an abnormality in the parents that causes the impairment to develop within the child.
In the United States the number of children a person has had is related to their risk of obesity. A woman's risk of obesity increases by 7% per child, while a man's risk increases by 4% per child. [24] This could be partly explained by the fact that having dependent children decreases physical activity in Western parents. [25]
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