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  2. Aldosterone - Wikipedia

    en.wikipedia.org/wiki/Aldosterone

    Aldosterone release causes sodium and water retention, which causes increased blood volume, and a subsequent increase in blood pressure, which is sensed by the baroreceptors. [39] To maintain normal homeostasis these receptors also detect low blood pressure or low blood volume, causing aldosterone to be released.

  3. Adrenal cortex - Wikipedia

    en.wikipedia.org/wiki/Adrenal_cortex

    The outermost layer, the zona glomerulosa is the main site for the production of aldosterone, a mineralocorticoid. The synthesis and secretion of aldosterone are mainly regulated by the renin–angiotensin–aldosterone system. The zona glomerulosa cells express a specific enzyme aldosterone synthase (also known as CYP11B2).

  4. Adrenal gland - Wikipedia

    en.wikipedia.org/wiki/Adrenal_gland

    Angiotensin II and extracellular potassium are the two main regulators of aldosterone production. [19] The amount of sodium present in the body affects the extracellular volume, which in turn influences blood pressure. Therefore, the effects of aldosterone in sodium retention are important for the regulation of blood pressure. [28] Glucocorticoids

  5. Angiotensin - Wikipedia

    en.wikipedia.org/wiki/Angiotensin

    Angiotensin also stimulates the release of aldosterone from the adrenal cortex to promote sodium retention by the kidneys. An oligopeptide , angiotensin is a hormone and a dipsogen . It is derived from the precursor molecule angiotensinogen, a serum globulin produced in the liver .

  6. Adrenocortical hormone - Wikipedia

    en.wikipedia.org/wiki/Adrenocortical_hormone

    The immune system specifically targets the cells of the adrenal cortex and destroys them, but Addison's disease can also be caused by a severe infection such as tuberculosis. Some symptoms include hypoglycemia and decreased blood sodium levels and increased blood potassium levels caused by a deficiency of aldosterone. These electrolyte ...

  7. Mineralocorticoid receptor - Wikipedia

    en.wikipedia.org/wiki/Mineralocorticoid_receptor

    The mineralocorticoid receptor (or MR, MLR, MCR), also known as the aldosterone receptor or nuclear receptor subfamily 3, group C, member 2, (NR3C2) is a protein that in humans is encoded by the NR3C2 gene that is located on chromosome 4q31.1-31.2. [5] MR is a receptor with equal affinity for mineralocorticoids and glucocorticoids.

  8. Is a cracked egg ever safe to eat? What you must know - AOL

    www.aol.com/cracked-egg-ever-safe-eat-100041198.html

    "That membrane protects [the egg] from bacteria," she said. "You can just crack the egg into a small bowl, cover it with plastic wrap or a lid, and use it within two days – making sure you cook ...

  9. Glucocorticoid remediable aldosteronism - Wikipedia

    en.wikipedia.org/wiki/Glucocorticoid_remediable...

    It selectively stimulates secretion of aldosterone. The secretion of aldosterone has a diurnal rhythm. Control of aldosterone release from the adrenal cortex: [citation needed] The role of the renin–angiotensin system: Angiotensin is involved in regulating aldosterone and is the core regulator. Angiotensin II acts synergistically with potassium.