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A human adult exhibiting voluntary control over his iris muscles, where he can cause his pupil to dilate and constrict on command. A dilation response , is the widening of the pupil and may be caused by adrenaline; anticholinergic agents; stimulant drugs such as MDMA, cocaine, and amphetamines; and some hallucinogenics (e.g. LSD). [3]
He attempted to perform this surgery a second time, but the patient did not survive. [81] Also during the 18th century, an English physician named Michael Underwood reported the symptoms associated with hydrocephalus. He described vomiting, fever, dilated pupils, sensitivity to light, and diplopia.
Both pupils are dilated (mydriasis) because of hyper-activation of the sympathetic system caused by the psychedelic drug LSD Mydriasis with the pupil dilated to 9mm in diameter as a result of dim lighting. Most individuals are not able to achieve pupillary dilation to this extent without mydriatic agents
Mild symptoms may consist of increased heart rate, shivering, sweating, dilated pupils, myoclonus (intermittent jerking or twitching), as well as hyperreflexia (overresponsive reflexes). [6] Many of these symptoms may be side effects of the drug or drug interaction causing excessive levels of serotonin rather than an effect of elevated ...
The skull bone behind Hermansen’s right eye was gone. In its place was a hole, allowing her brain to press against the eye, which explained the pulsating, said Dr. Netanel Ben-Shalom, a ...
Situational syncope is often triggered by urination, swallowing, or coughing. [2] Carotid sinus syncope is due to pressure on the carotid sinus in the neck. [2] The underlying mechanism involves the nervous system slowing the heart rate and dilating blood vessels, resulting in low blood pressure and thus not enough blood flow to the brain. [2]
Often, patients can only recognize their prodrome symptoms when they get to the pain phase and look back, Singh says. During a prodrome period, the Mayo Clinic and American Migraine Foundation say ...
The term was first discovered in 1791 by Mondini when he was completing a temporal bone dissection. It was then defined by Valvassori and Clemis as a vestibular aqueduct that is greater than or equal to 2.0 mm at the operculum and/or greater than or equal to 1.0 mm at the midpoint. [ 2 ]