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Degenerative suspensory ligament desmitis, commonly called DSLD, also known as equine systemic proteoglycan accumulation (ESPA), is a systemic disease of the connective tissue of the horse and other equines. It is a disorder akin to Ehlers–Danlos syndrome being researched in multiple horse breeds.
The callus formed due to splint bone injury can become large and put pressure on the suspensory ligament. Bone heals by formation of a callus over the defective area. Speed and quality of healing is directly related to the blood supply and fracture stability. Rest is required immediately following injury to reduce movement of the fracture site.
Tendinosis is chronic degeneration of a tendon without inflammation. It is caused by repetitive microtrauma and aging. [67] Tendinosis is characterized by painful thickening and structural changes of the tendon. [68] Tendinosis predisposes horses for Tendinitis and catastrophic ruptured tendons. [65] [69] Desmitis is inflammation of a ligament.
Degenerative joint disease (DJD), such as bone spavin, ringbone, omarthritis; Inflammatory joint disease such as Carpitis (sprained knee), osselets; Bucked shins; Curb; Degenerative suspensory ligament desmitis (DSLD), and sprains of the suspensory ligament; Fractures; Locked kneecap (delayed patellar release or intermittent upward fixation of ...
The Akhal-Teke is one of many light riding horse breeds that may be prone to cervical vertebral malformation (CVM), commonly called Wobbler syndrome, [61] and to Degenerative suspensory ligament desmitis (DSLD). [62] These conditions are seen in a number of other breeds, including the Thoroughbred. There is likely a genetic component to Wobbler ...
Degenerative suspensory ligament desmitis is a similar condition seen in many breeds of horses. [169] It was originally notated in the Peruvian Paso and thought to be a condition of overwork and older age, but it is being recognized in all age groups and all activity levels. It has been noted in newborn foals.
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Degenerative suspensory ligament desmitis; Dermatoosteolysis, Kirghizian type; Desmin-related myofibrillar myopathy; Distal hereditary motor neuropathy type V; Distal spinal muscular atrophy type 2; Distichiasis, congenital heart defects and mixed peripheral vascular anomalies; DOCK8 deficiency; Dunnigan familial partial lipodystrophy