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The treatment of equine lameness is a complex subject. Lameness in horses has a variety of causes, and treatment must be tailored to the type and degree of injury, as well as the financial capabilities of the owner. Treatment may be applied locally, systemically, or intralesionally, and the strategy for treatment may change as healing progresses.
Degenerative suspensory ligament desmitis, commonly called DSLD, also known as equine systemic proteoglycan accumulation (ESPA), is a systemic disease of the connective tissue of the horse and other equines. It is a disorder akin to Ehlers–Danlos syndrome being researched in multiple horse breeds.
Joints should be palpated for pain, effusion of joint pouches, thickening of the joint capsule, and checked for range of motion. Major ligaments and tendons, such as the superficial and deep digital flexor tendons, inferior check ligament, suspensory ligament, and distal sesamoidean ligaments, should also be palpated along their entire length.
Neurectomy is also used in equine medicine, primarily for cases of persistent lameness that is non-responsive to other forms of treatment. It is most commonly used for animals with navicular syndrome and suspensory ligament desmitis.
The suspensory apparatus consists of the suspensory ligament, the check ligament, the deep digital flexor tendon, the superficial flexor tendon, the common digital extensor tendon and the sesamoid bones. [11] Horses use a group of ligaments, tendons and muscles known as the stay apparatus to "lock" major joints in the limbs, allowing them to ...
Injury to this ligament is an important cause of lameness in performance horses. The suspensory is a modified muscle, the equine equivalent of the interosseous muscle, which contains both tendon fibers and residual muscle fibers. [1] Interosseous ligaments: connect the cannon bone to each splint bone.
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