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  2. Your Gout Guide: From Symptoms to Treatment - AOL

    www.aol.com/gout-guide-symptoms-treatment...

    If you have frequent gout flares, gout associated with tophi, or joint damage, your provider may prescribe medication to lower uric acid levels. These can help prevent gout flares and further ...

  3. Managing Out-of-Control Chronic Gout: Going Beyond Oral ...

    www.aol.com/lifestyle/managing-control-chronic...

    The post Managing Out-of-Control Chronic Gout: Going Beyond Oral Treatments appeared first on Reader's Digest. Do you sometimes have severe, unexplained pain in your joints, particularly in your ...

  4. Pyrazinamide - Wikipedia

    en.wikipedia.org/wiki/Pyrazinamide

    The most common (roughly 1%) side effect of pyrazinamide is joint pains (arthralgia), but this is not usually so severe that patients need to stop taking it. [10] [11] Pyrazinamide can precipitate gout flares by decreasing renal excretion of uric acid. [12] The most dangerous side effect of pyrazinamide is hepatotoxicity, which is dose-related ...

  5. Allopurinol - Wikipedia

    en.wikipedia.org/wiki/Allopurinol

    Common side effects when used orally include itchiness and rash. [7] Common side effects when used by injection include vomiting and kidney problems. [7] While not recommended historically, starting allopurinol during an attack of gout appears to be safe. [8] [9] In those already on the medication, it should be continued even during an acute ...

  6. Colchicine - Wikipedia

    en.wikipedia.org/wiki/Colchicine

    Colchicine is a medication used to prevent and treat gout, [3] [4] to treat familial Mediterranean fever [5] and Behçet's disease, [6] and to reduce the risk of myocardial infarction. [7] The American College of Rheumatology recommends colchicine, nonsteroidal anti-inflammatory drugs (NSAIDs) or steroids in the treatment of gout.

  7. Probenecid - Wikipedia

    en.wikipedia.org/wiki/Probenecid

    In gout, probenecid competitively inhibits the reabsorption of uric acid through the organic anion transporter (OAT) at the proximal tubules. This leads to preferential reabsorption of probenecid back into plasma and excretion of uric acid in urine, [ 6 ] thus reducing blood uric acid levels and reducing its deposition in various tissues.

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