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  2. Complement factor I - Wikipedia

    en.wikipedia.org/wiki/Complement_factor_I

    Complement factor I, also known as C3b/C4b inactivator, is a protein that in humans is encoded by the CFI gene. Complement factor I (factor I) is a protein of the complement system , first isolated in 1966 in guinea pig serum , [ 5 ] that regulates complement activation by cleaving cell-bound or fluid phase C3b and C4b. [ 6 ]

  3. Complement component 3 - Wikipedia

    en.wikipedia.org/wiki/Complement_component_3

    First, the proteolytic component of the convertase, Bb, is removed by complement regulatory proteins having decay-accelerating factor (DAF) activity. Next, C3b is broken down progressively to first iC3b, then C3c + C3dg, and then finally C3d. Factor I is the protease cleaves C3b but requires a cofactor (e.g Factor H, CR1, MCP or C4BP) for activity.

  4. C3-convertase - Wikipedia

    en.wikipedia.org/wiki/C3-convertase

    Properdin (Factor P) is the only known positive regulator of complement activation that stabilizes the alternative C3 convertase (C3bBb). Properdin deficient individuals are sensitive to pyogenic infections. Properdin also promotes association of C3b with Factor B and thus it inhibits the Factor H mediated cleavage of C3b by Factor I. [6]

  5. Complement fixation test - Wikipedia

    en.wikipedia.org/wiki/Complement_fixation_test

    The complement fixation test is an immunological medical test that can be used to detect the presence of either specific antibody or specific antigen in a patient's serum, based on whether complement fixation occurs. It was widely used to diagnose infections, particularly with microbes that are not easily detected by culture methods, and in ...

  6. Complement deficiency - Wikipedia

    en.wikipedia.org/wiki/Complement_deficiency

    Complement deficiency is an immunodeficiency of absent or suboptimal functioning of one of the complement system proteins. [4] Because of redundancies in the immune system, many complement disorders are never diagnosed. Some studies estimate that less than 10% are identified. [5]

  7. Classical complement pathway - Wikipedia

    en.wikipedia.org/wiki/Classical_complement_pathway

    Complement is responsible for immune inflammatory response in adipose tissues which has been implicated in the development of obesity. [8] Obesity in turn results in an abnormally high level of complement activation via production of the C1 component of the classical pathway, which can lead to tissue inflammation and eventually insulin ...

  8. Alternative complement pathway - Wikipedia

    en.wikipedia.org/wiki/Alternative_complement_pathway

    The classical and alternative complement pathways. Alternative pathway. (Some labels are in Polish.) The alternative pathway is a type of cascade reaction of the complement system and is a component of the innate immune system, a natural defense against infections.

  9. Complement system - Wikipedia

    en.wikipedia.org/wiki/Complement_system

    The complement system, also known as complement cascade, is a part of the humoral, innate immune system and enhances (complements) the ability of antibodies and phagocytic cells to clear microbes and damaged cells from an organism, promote inflammation, and attack the pathogen's cell membrane. [1]