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Randle suggested that the term fatty acid syndrome would be appropriate to apply to the biochemical syndrome resulting from the high concentration of fatty acids and the relationship to abnormalities of carbohydrate metabolism, including starvation, diabetes and Cushing’s syndrome. [4]
Carbohydrate metabolism is the whole of the biochemical processes responsible for the metabolic formation, breakdown, and interconversion of carbohydrates in living organisms. Carbohydrates are central to many essential metabolic pathways . [ 1 ]
On average, the starvation response of the individuals after isolation was a 750-kilojoule (180-kilocalorie) reduction in daily total energy expenditure. 250 kJ (60 kcal) of the starvation response was explained by a reduction in fat-free mass and fat mass. An additional 270 kJ (65 kcal) was explained by a reduction in fidgeting. The remaining ...
In ruminants, because dietary carbohydrates tend to be metabolized by rumen organisms, gluconeogenesis occurs regardless of fasting, low-carbohydrate diets, exercise, etc. [3] In many other animals, the process occurs during periods of fasting, starvation, low-carbohydrate diets, or intense exercise.
Frederick Madison Allen (March 16, 1879 – April 14, 1957) was an American physician who is best remembered for his carbohydrate-restricted low-calorie diet for sufferers of diabetes mellitus. He was known for developing the "starvation diet" as a treatment. [1]
Fatty acid metabolism consists of various metabolic processes involving or closely related to fatty acids, a family of molecules classified within the lipid macronutrient category. These processes can mainly be divided into (1) catabolic processes that generate energy and (2) anabolic processes where they serve as building blocks for other ...
George F. Cahill Jr. (July 7, 1927 – July 30, 2012) was an American scientist who significantly advanced the diabetes mellitus research of the 20th century. [1] He focused on metabolic research, especially concerning human glucose metabolism in diabetic and normal conditions; he also investigated the effect of hunger and fasting on metabolic pathways and ketose processes.
Ketogenesis takes place in the setting of low glucose levels in the blood, after exhaustion of other cellular carbohydrate stores, such as glycogen. [10] It can also take place when there is insufficient insulin (e.g. in type 1 (and less commonly type 2) diabetes), particularly during periods of "ketogenic stress" such as intercurrent illness. [4]