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The biggest concerns about pulmonary fibrosis and the increase of respiratory follow-up after COVID-19 are expected to be solved in the near future. Older age with decreased lung function and/or preexisting comorbidities, such as diabetes, cardiovascular disease, hypertension, and obesity increase the risk of developing fibrotic lung ...
Fibrosis can affect one or both of the two layers of tissue forming the pleura—the visceral pleura adjacent to the lung and the parietal pleura adjacent to the ribcage. The term fibrothorax implies severe fibrosis affecting both the visceral and the outer (parietal) pleura, fusing the lung to the chest wall. [2]
The median survival in idiopathic pulmonary fibrosis is 3-3.5 years. In those who receive a lung transplant, the medial survival in idiopathic pulmonary fibrosis is 5.2 years, as compared to 6.7 years in those with other types of ILD. [8] ILD is associated with a 3-fold increased risk of lung cancer. [8]
In COVID-19, the arterial and general tissue oxygen levels can drop without any initial warning.The chest x-ray may show diffuse pneumonia.Cases of silent hypoxia with COVID-19 have been reported for patients who did not experience shortness of breath or coughing until their oxygen levels had depressed to such a degree that they were at risk of acute respiratory distress (ARDS) and organ failure.
An overall definition of post-COVID conditions (PCC) can be described as a range of symptoms that can last for weeks or months. [83] Long COVID can be present in anyone who has contracted COVID-19 at some point; typically, it is more commonly found in those who had severe illness due to the virus. [83] [84]
In the late 2000s, David Smadja and his team proposed that the pulmonary endothelium is an active organ in pulmonary diseases, particularly in pulmonary hypertension (PH) and idiopathic pulmonary fibrosis (IPF), marked by the destruction of alveolar architecture.
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