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Neurological collapse can occur within a short period, [12] with rapid onset of dilating pupils, loss of eye movement, unconsciousness, and respiratory failure. [11] Failure of the brain stem frequently occurs between two and five minutes after the second impact, [1] [2] [11] and death can follow shortly. [8]
The ultimate consequence of cytotoxic edema is the oncotic death of neurons. [1] The swelling of the individual cells of the brain is the main distinguishing characteristic of cytotoxic edema, as opposed to vasogenic edema, wherein the influx of fluid is typically seen in the interstitial space rather than within the cells themselves. [20]
These factors cause the brain to swell with fluid, resulting in severe impairment. [15] If the swelling is untreated, it causes death by brain herniation. [4] The brain swelling is likely a result of vasogenic edema, the penetration of the blood–brain barrier by fluids. [16] This process has been observed in MRI studies.
An increase in pressure, most commonly due to head injury leading to intracranial hematoma or cerebral edema, can crush brain tissue, shift brain structures, contribute to hydrocephalus, cause brain herniation, and restrict blood supply to the brain. [13] It is a cause of reflex bradycardia. [14]
Closed-head injuries can range from mild injuries to debilitating traumatic brain injuries and can lead to severe brain damage or death. Common closed-head injuries include: [5] concussion – a head injury resulting in temporary dysfunction of normal brain function. Almost half of the total concussions reported each year are sports-related [5]
When Olivia Palermo, 19, first noticed a twitch in her finger, she chalked it up to dehydration. But as her symptoms worsened, doctors told her she had a mental health condition—not a serious ...
The cause of death: cerebral edema, a swelling of the brain brought on after Lee had taken too much aspirin." It also mentioned that toward the end of his life, Lee had suffered "several public ...
Another factor in secondary injury is loss of cerebral autoregulation, the ability of the brain's blood vessels to regulate cerebral blood flow. [5] Other factors in secondary damage are breakdown of the blood–brain barrier, edema, ischemia and hypoxia. [15] Ischemia is one of the leading causes of secondary brain damage after head trauma. [9]