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Black silver horse exhibiting strongly diluted long hair with darker roots and flat gray, dappled body color. The silver or silver dapple (Z) gene is a dilution gene that affects the black base coat color and is associated with Multiple Congenital Ocular Abnormalities.
The horse has a "visual streak", or an area within the retina, linear in shape, with a high concentration of ganglion cells (up to 6100 cells/mm 2 in the visual streak compared to the 150 and 200 cells/mm 2 in the peripheral area). [12] Horses have better acuity when the objects they are looking at fall in this region.
"Moon blindness" on an Icelandic horse Advanced stage of disease Advanced stage of disease ERU on an Icelandic horse. Equine recurrent uveitis (ERU) – also known as moon blindness, recurrent iridocyclitis, or periodic ophthalmia [1] – is an acute, nongranulomatous inflammation of the uveal tract of the eye, occurring commonly in horses of all breeds, worldwide.
A horse's genotype may be lp/lp (homozygous recessive), Lp/lp (heterozygous), or Lp/Lp (homozygous dominant). Horses without a dominant Lp gene do not exhibit leopard-complex traits, and cannot produce offspring with the Lp gene unless it is contributed by the other parent. Such horses are termed "non-characteristic" among Appaloosa horse ...
The impact of snow blindness extended to animals as well; the expedition’s horses suffered from the condition. Lawrence Oates proposed dyeing the horses’ forelocks as a preventative measure, and they were also equipped with tassels over their eyes for protection. Similarly, mules were provided with canvas snow goggles, demonstrating the ...
Historical reenactment of a Sasanian-era cataphract, complete with a full set of scale armour for the horse. The rider is covered by extensive mail armour.. A cataphract was a form of armoured heavy cavalry that originated in Persia and was fielded in ancient warfare throughout Eurasia and Northern Africa.
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Horses cannot pass the disease among themselves; that is, one horse cannot contract the disease from another infected horse. The horse is a dead-end, or aberrant, host of the parasite. Although all horses are believed to be susceptible to EPM the disease is usually found in younger horses typically around three to six years of age. [2]