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Canna indica was also part of this famine food. As a consequence, the plant became known as a food crop. Today, the rhizomes are processed to starch, vermicelli, white wine, and ethanol. Due to the use as a food crop and relatively low diseases and pests pressure, Canna indica has become a characteristic crop for large-scale cultivation in ...
The name Canna originates from the Latin word for a cane or reed. [13] Canna indica, commonly called achira in Latin America, has been cultivated by Native Americans in tropical America for thousands of years, and was one of the earliest domesticated plants in the Americas. The starchy root is edible. [14]
A dried cannabis flower. The short-term effects of cannabis are caused by many chemical compounds in the cannabis plant, including 113 [clarification needed] different cannabinoids, such as tetrahydrocannabinol, and 120 terpenes, [1] which allow its drug to have various psychological and physiological effects on the human body.
High blood pressure is caused by the force of blood flow in the arteries being too high. The DASH diet includes heart-healthy foods that lower blood pressure. 21 foods that lower blood pressure ...
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This amendment reflects an increasing bipartisan acknowledgment of the potential therapeutic uses of cannabis and the significance of state-level policymaking in this area. Classification In the U.S., the National Institute on Drug Abuse defines medical cannabis as "using the whole, unprocessed marijuana plant or its basic extracts to treat ...
Cannabinoid production starts when an enzyme causes geranyl pyrophosphate and olivetolic acid to combine and form CBGA. Next, CBGA is independently converted to either CBG, THCA, CBDA or CBCA by four separate synthase, FAD-dependent dehydrogenase enzymes. There is no evidence for enzymatic conversion of CBDA or CBD to THCA or THC.
In vitro, CBG has identified pharmacodynamic actions and its mechanism of action appears to be from interactions with multiple targets. [1] [2] [3]CBG is a weak ligand of the cannabinoid CB 1 and CB 2 receptors with affinities (K i) of 380–2,600 nM and 153–3,460 nM, respectively.