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In comparison to eukaryotes, the intracellular features of the bacterial cell are extremely simple. Bacteria do not contain organelles in the same sense as eukaryotes. Instead, the chromosome and perhaps ribosomes are the only easily observable intracellular structures found in all bacteria. There do exist, however, specialized groups of ...
When an intracellular parasite goes to enter a host cell, it is particular about the type of host cell. This is because most intracellular parasites are able to infect only a few different cell types. [21] Viruses use a number of host receptors to gain entry to the cell, usually by causing endocytosis. [7]
Intracellular bacteria are bacteria that have the capability to enter and survive within the cells of the host organism. [1] These bacteria include many different pathogens that live in the cytoplasm and nuclei of the host cell's they inhabit. Two examples of intracellular pathogenic bacteria are Mycobacterium tuberculosis and also Toxoplasma ...
Size Reference Flatworm animal: Eucestoda: Tapeworm: length (maximum) 25 m: Eucestoda: Nematode animal: Loa loa: Loa loa: length (female) 20–70 mm: Loa loa: Arthropod animal: Cymothoa exigua: Tongue-eating louse: length (female) 8–29 mm: Cymothoa exigua: Nematode animal: Enterobius: Pinworm: length (female) 8–13 mm: Pinworm (parasite ...
Spiral bacteria are another major bacterial cell morphology. [2] [30] [31] [32] Spiral bacteria can be sub-classified as spirilla, spirochetes, or vibrios based on the number of twists per cell, cell thickness, cell flexibility, and motility. [33] Bacteria are known to evolve specific traits to survive in their ideal environment. [34]
Chlamydia trachomatis is a gram-negative bacterium that can replicate exclusively within a host cell, making it an obligate intracellular pathogen. [3] Over the course of its life cycle, C. trachomatis takes on two distinct forms to facilitate infection and replication.
[15] [16] [17] Because the organism is an obligate intracellular parasite, it lacks many necessary genes for independent survival, causing difficulty in culturing the organism. The complex and unique cell wall that makes members of the genus Mycobacterium difficult to destroy is also the reason for its extremely slow replication rate.
While in the human host, the parasite spends most of its life cycle within hepatic cells and erythrocytes (in contrast to T. brucei which remains extracellular). As a result of its mainly intracellular niche, parasitized host cells which display parasite proteins must be modified to prevent destruction by the host immune defenses.