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Superior mesenteric artery compressing the duodenum, featuring the superior mesenteric artery syndrome. Superior mesenteric artery (SMA) syndrome is a gastro-vascular disorder in which the third and final portion of the duodenum is compressed between the abdominal aorta (AA) and the overlying superior mesenteric artery.
ISMAD primarily manifests through a sudden onset of pain, which can vary in location and intensity. [2] The nature of the pain and its location can provide clues to the diagnosis of ISMAD. [2] The types of pain reported in ISMAD cases include: Abdominal pain: This is the most common symptom, reported in 55.8% of cases. [2]
In human anatomy, the superior mesenteric artery (SMA) is an artery which arises from the anterior surface of the abdominal aorta, just inferior to the origin of the celiac trunk, and supplies blood to the intestine from the lower part of the duodenum through two-thirds of the transverse colon, as well as the pancreas.
Abdominal angina often has a one-year delay between symptoms and treatment, leading to complications like malnutrition or bowel infarction. Abdominal angina is more prevalent in females with a 3:1 ratio, and the average age of onset is 60 years. Abdominal angina was first described by Dr. Baccelli in 1918 as lower abdominal pain after eating.
The signs and symptoms of NCS are all derived from the outflow obstruction of the left renal vein. The compression causes renal vein hypertension, leading to hematuria (which can lead to anemia) [4] and abdominal pain (classically left flank or pelvic pain). [5] The abdominal pain may improve or worsen depending on positioning. [5]
Anemia of chronic disease: AODM Adult onset diabetes mellitus: AOS Apraxia of speech: APA Aldosterone-producing adenoma: APS Antiphospholipid syndrome: ARBD Alcohol-related birth defects: ARD Adult Refsum disease: ARDS Acute respiratory distress syndrome: ARND Alcohol-related neurodevelopmental disorder: ARM Anorectal malformation: AS ...
Often Segmental Arterial Mediolysis is diagnosed after clinical presentation with symptoms as above followed by CT angiogram or MRI demonstrating aneurysm(s). [4] The gold standard method for confirming the diagnosis is surgical resection of the affected area of blood vessel followed by histologic investigation under a microscope. [ 3 ]
The disease's symptoms can range from catastrophic neurological impairment to an asymptomatic condition brought on by impalpable pulses or bruits. [7] Non-specific features include mild anemia, myalgia, arthralgia, weight loss, malaise, night sweats, and fever. [8] Giant cell arteritis (GCA) is the most common type of systemic vasculitis in adults.
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