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Experiments with pharmacological agents that cause depletion of monoamines have shown that this depletion does not cause depression in healthy people. [56] [57] Another problem that presents is that drugs that deplete monoamines may actually have antidepressants properties.
Magnesium can not be produced by the human body, and can only be obtained through dietary means. [3] When the amount of magnesium levels in the blood falls below the normal level (1.3 to 2.1 mEq/L), a person is experiencing hypomagnesia, or magnesium deficiency. [2]
In addition to post-treatment relapse, depressive symptoms can even recur in the course of long-term therapy (tachyphylaxis). Also, currently available antidepressants all elicit undesirable side-effects, and new agents should be divested of the distressing side-effects of both first and second-generation antidepressants. [6]
However, vitamin B12 supplements could still have a place in your weight loss treatment plan. A vitamin B12 deficiency — which, as mentioned, those with overweight or obesity are more at risk ...
The pharmacology of antidepressants is not entirely clear.. The earliest and probably most widely accepted scientific theory of antidepressant action is the monoamine hypothesis (which can be traced back to the 1950s), which states that depression is due to an imbalance (most often a deficiency) of the monoamine neurotransmitters (namely serotonin, norepinephrine and dopamine). [1]
Some antidepressants, including commonly prescribed ones from the class known as SSRIs (selective serotonin reuptake inhibitors), can cause sexual side effects such as erectile dysfunction ...
What’s more, once a person has tried two different antidepressants, stayed with each one for long enough to feel the effects, and doesn’t get relief, their depression is considered treatment ...
On the other hand, significantly more patients drop off from the antidepressant treatment than from psychotherapy, likely because of the side effects of antidepressants. [106] Successful psychotherapy appears to prevent the recurrence of depression even after it has been terminated or replaced by occasional "booster" sessions.
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