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Other important factors for risk assessment include the likelihood that a snake will bite, the quantity of venom delivered with the bite, the efficiency of the delivery mechanism, and the location of a bite on the body of the victim. Snake venom may have both neurotoxic and hemotoxic properties. There are about 600 venomous snake species in the ...
Two main hemotoxic effects are caused by rattlesnake venom. First, zinc-containing metalloproteases act upon capillary endothelial cells to inhibit platelet aggregation and hemorrhage. [ 14 ] Second, the platelet antagonist crotalin creates a severe bleeding effect as it binds to the surface proteins, blocking aggregation.
It is thought that this was the result of descent from a common venom-producing squamate ancestor; the hypothesis was described simply as the "venom clade" when first proposed to the scientific community. [2] The venom clade included Anguidae for phylogenetic reasons and adopted a previously suggested clade name: Toxicofera. [14]
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Vipera berus - Venom delivery apparatus. Snake venom is a highly toxic saliva [1] containing zootoxins that facilitates in the immobilization and digestion of prey. This also provides defense against threats. Snake venom is usually injected by unique fangs during a bite, though some species are also able to spit venom. [2]
Average venom yield per bite is 37 mg and a maximum yield of 97 mg. [164] Bites from red-bellied black snake are rarely life-threatening due to the snake usually choosing to inject little venom toxin, but are still in need of immediate medical attention. Rate of envenomation is 40–60%, but the untreated mortality rate is less than 1%.
Common names: Hopi rattlesnake, [2] Arizona prairie rattlesnake, prairie rattlesnake. [3]Crotalus viridis nuntius is a venomous pit viper subspecies [4] native primarily to the desert plateau of the northeastern portion of the American state of Arizona, but also ranges into northwestern New Mexico.
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