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Frontal bossing is the development of an unusually pronounced forehead which may also be associated with a heavier than normal brow ridge. It is caused by enlargement of the frontal bone , often in conjunction with abnormal enlargement of other facial bones , skull , mandible , and bones of the hands and feet.
Compensatory growth occurs forward at the coronal suture and backward at the lambdoid suture giving respectively a prominent forehead, called frontal bossing, and a prominent back portion of the head, called coning. [10] [11] This is the most common form of craniosynostosis. [13]
Parrot's sign, [3] also known as 'Parrot's nodes' [4] and 'Parrot's bosses', [5] [6] refers to the bony growth noted at autopsy by Marie Jules Parrot and Jonathan Hutchinson on the skulls of children with congenital syphilis (CS) in the 19th century. [2] [7] Later publications also describe it as the frontal bossing that presents in the late ...
Kagami-Ogata syndrome is a rare genetic disease that is caused by mutations on Maternal chromosome 14 or by paternal UPD(14). [1] The main signs of this disease are: polyhydramnios, narrow bell-shaped thorax, coat-hanger-like ribs, abdominal wall defect, enlarged placenta. [2]
However, if diagnosis of congenital syphilis is delayed until Hutchinson’s triad is noted–among other signs and symptoms, such as nasal cartilage destruction (saddle nose), frontal bossing, joint swelling (Clutton joints), tibial thickening (Saber shins), hard palate defect–the damage is irreversible. [4]
Early skeletal deformities can arise in infants such as soft, thinned skull bones – a condition known as craniotabes, [15] [16] which is the first sign of rickets; skull bossing may be present and a delayed closure of the fontanelles. Young children may have bowed legs and thickened ankles and wrists; [17] older children may have knock knees ...
PDP has a number of visible signs. Most important clinical features are: pachydermia (thickening and wrinkling of the skin), furrowing of the face and scalp, periostosis (swelling of periarticular tissue and shaggy periosteal new bone formation of long bones) and digital clubbing (enlargement of fingertips). [1]
Early recognition of this injury is crucial for survival. Infants who have experienced a difficult operative delivery or are suspected to have a SGH require ongoing monitoring including frequent vital signs (minimally every hour), and serial measurements of hematocrits and their occipital frontal circumference, which increases 1 cm with each 40 mL of blood deposited into the subgaleal space.