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Agonal heart rhythm is usually ventricular in origin. Occasional P waves and QRS complexes can be seen on the electrocardiogram. The complexes tend to be wide and bizarre in morphological appearance. [1] Clinically, an agonal rhythm is regarded as asystole and should be treated equivalently, with cardiopulmonary resuscitation and administration ...
Accelerated idioventricular rhythm; Accessory pathway; Adams–Stokes syndrome; Agonal heart rhythm; Andersen–Tawil syndrome; Pacemaker failure; Ashman phenomenon; Asystole; Atrial fibrillation; Atrial fibrillation with rapid ventricular response; Atrial flutter; Atrial tachycardia; Atrioventricular block; Atrioventricular dyssynchrony
[6] The escape rhythm typically originates in the ventricles, producing a wide complex escape rhythm. Third-degree heart block may also be congenital and has been linked to the presence of lupus in the mother. [7] It is thought that maternal antibodies may cross the placenta and attack the heart tissue during gestation. The cause of congenital ...
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Return of spontaneous circulation (ROSC) is the resumption of a sustained heart rhythm that perfuses the body after cardiac arrest. It is commonly associated with significant respiratory effort. Signs of return of spontaneous circulation include breathing, coughing, or movement and a palpable pulse or a measurable blood pressure.
Ventricular fibrillation (V-fib or VF) is an abnormal heart rhythm in which the ventricles of the heart quiver. [2] It is due to disorganized electrical activity. [2] Ventricular fibrillation results in cardiac arrest with loss of consciousness and no pulse. [1]
They conduct cardiac action potentials more quickly and efficiently than any of the other cells in the heart's electrical conduction system. [4] Purkinje fibers allow the heart's conduction system to create synchronized contractions of its ventricles, and are essential for maintaining a consistent heart rhythm. [5]
Ventricular escape beats occur when the rate of electrical discharge reaching the ventricles (normally initiated by the heart's sinoatrial node (SA node), transmitted to the atrioventricular node (AV node), and then further transmitted to the ventricles) falls below the base rate determined by the rate of Phase 4 spontaneous depolarisation of ventricular pacemaker cells. [1]
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262 Neil Avenue # 430, Columbus, Ohio · Directions · (614) 221-7464