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The first article to propose Morgellons as a new disease in a scientific journal was a review article co-authored by members of the MRF and published in 2006 by the American Journal of Clinical Dermatology. [25] A 2006 article in the San Francisco Chronicle reported, "There have been no clinical studies" of Morgellons disease. [25]
Pseudoxanthoma elasticum (PXE) is a genetic disease that causes mineralization of elastic fibers in some tissues. The most common problems arise in the skin and eyes, and later in blood vessels in the form of premature atherosclerosis. [2] [3] [4] PXE is caused by autosomal recessive mutations in the ABCC6 gene on the short arm of chromosome 16 ...
A skin biopsy for the measurement of epidermal nerve fiber density is an increasingly common technique for the diagnosis of small fiber peripheral neuropathy. [13] Physicians can biopsy the skin with a 3-mm circular punch tool and immediately fix the specimen in 2% paraformaldehyde lysine-periodate or Zamboni's fixative. [20]
Treatment: Ringworm can usually be treated with antifungal creams, lotions, or powders applied to the skin for two to four weeks, according to the Centers for Disease Control and Prevention (CDC ...
In the earlier stages of actinic elastosis, elastic fiber proliferation can be seen in the dermis. As the condition becomes more established, the collagen fibers of the papillary dermis and reticular dermis become increasingly replaced by thickened and curled fibers that form tangled masses and appear basophilic under routine haematoxylin and eosin staining.
Cutis laxa is associated with deficient or absent elastin fibers in the extracellular matrix. [6] This can be related to decreased elastin synthesis or structural defects in the extracellular matrix. [7] Cutis laxa may be caused by mutations in the genes: ELN, [8] ATP6V0A2, [9] ATP7A, [10] FBLN4, [11] FBLN5, [12] and PYCR1. [13]
seek treatment during middle-age, although PN can occur at any age. have a history of chronic severe pruritus. have a significant medical history for unrelated conditions. develop liver or kidney dysfunctions. develop secondary skin infections. have a personal or family history of atopic dermatitis. have other autoimmune disorders.
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