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Chains of S. pyogenes bacteria (orange) at 900× magnification Gram stain of Streptococcus pyogenes. Unlike most bacterial pathogens, S. pyogenes only infects humans. Thus, zoonotic transmission from an animal (or animal products) to a human is rare. [8] S. pyogenes typically colonizes the throat, genital mucosa, rectum, and skin. Of healthy ...
Group A streptococcal infections are a number of infections with Streptococcus pyogenes, a group A streptococcus (GAS). [1] S. pyogenes is a species of beta-hemolytic Gram-positive bacteria that is responsible for a wide range of infections that are mostly common and fairly mild. If the bacteria enters the bloodstream, the infection can become ...
The speB and speJ genes are located in the core bacterial chromosome of all strains of S. pyogenes. [3] [14] However, despite its presence and high levels of conservation in the nucleotide sequence, 25–40% of these strains do not express the SpeB toxin in significant amounts. [14] In contrast, speA, speC and speH-M are encoded by ...
Streptococcal pharyngitis, also known as streptococcal sore throat (strep throat), is pharyngitis (an infection of the pharynx, the back of the throat) caused by Streptococcus pyogenes, a gram-positive, group A streptococcus. [9] [10] Common symptoms include fever, sore throat, red tonsils, and enlarged lymph nodes in the front of the neck.
Toxic shock syndrome (TSS) is a condition caused by bacterial toxins. [1] Symptoms may include fever, rash, skin peeling, and low blood pressure. [1] There may also be symptoms related to the specific underlying infection such as mastitis, osteomyelitis, necrotising fasciitis, or pneumonia.
M protein is strongly anti-phagocytic and is the major virulence factor for group A streptococci (Streptococcus pyogenes). It binds to serum factor H, destroying C3-convertase and preventing opsonization by C3b. However plasma B cells can generate antibodies against M protein which will help in opsonization and further the destruction of the ...
The rash occurs as a result of capillary damage by exotoxins produced by S.pyogenes. [6] On darker-pigmented skin the rash may be hard to discern. [7] Scarlet fever develops in a small number of people who have strep throat or streptococcal skin infections. [1] The bacteria are usually spread by people coughing or sneezing. [1]
The 16S groups have been confirmed by whole genome sequencing (see figure). The important pathogens S. pneumoniae and S. pyogenes belong to the S. mitis and S. pyogenes groups, respectively, [29] while the causative agent of dental caries, Streptococcus mutans, is basal to the Streptococcus group.