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Sticky skin syndrome or acquired cutaneous adherence is a condition where the skin becomes sticky and objects may adhere to it. It is occasionally caused by the use of pharmaceutical drugs and chemotherapy drugs .
This is a shortened version of the twelfth chapter of the ICD-9: Diseases of the Skin and Subcutaneous Tissue. It covers ICD codes 680 to 709. The full chapter can be found on pages 379 to 393 of Volume 1, which contains all (sub)categories of the ICD-9. Volume 2 is an alphabetical index of Volume 1.
Epidermolysis bullosa acquisita, also known as acquired epidermolysis bullosa, is a longterm autoimmune blistering skin disease. [1] It generally presents with fragile skin that blisters and becomes red with or without trauma. [2] Marked scarring is left with thin skin, milia and nail changes. [3] It typically begins around age 50. [2]
Epidermolysis bullosa dystrophica or dystrophic EB (DEB) is an inherited disease affecting the skin and other organs. [1] [2] "Butterfly child" is the colloquial name for children born with the disease, as their skin is seen to be as delicate and fragile as the wings of a butterfly. [3]
This category reflects the organization of International Statistical Classification of Diseases and Related Health Problems, 10th Revision. Generally, diseases outlined within the ICD-10 codes L10–L14 within Chapter XII: Diseases of the skin and subcutaneous tissue should be included in this category.
SAPHO syndrome includes a variety of inflammatory bone disorders that may be associated with skin changes. These diseases share some clinical, radiologic, and pathologic characteristics. These diseases share some clinical, radiologic, and pathologic characteristics.
Erythroderma is an inflammatory skin disease with redness and scaling that affects nearly the entire cutaneous surface. [ 1 ] [ 2 ] This term applies when 90% or more of the skin is affected. In ICD-10 , a distinction is made between "exfoliative dermatitis" at L26, and "erythroderma" at L53.9.
The term "CYLD cutaneous syndrome" as applied to individuals with MFT1, BSS, or FC hair follicular tumors and carrying a hereditary CYLD mutation was first proposed by Rajan et al. in 2009. [6] Individuals with CCS generally develop increasing numbers of benign skin tumors beginning in their youth and continuing throughout most of their lives.