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The abducens nerve or abducent nerve, also known as the sixth cranial nerve, cranial nerve VI, or simply CN VI, is a cranial nerve in humans and various other animals that controls the movement of the lateral rectus muscle, one of the extraocular muscles responsible for outward gaze. It is a somatic efferent nerve.
Neuroinflammation is widely regarded as chronic, as opposed to acute, inflammation of the central nervous system. [5] Acute inflammation usually follows injury to the central nervous system immediately, and is characterized by inflammatory molecules, endothelial cell activation, platelet deposition, and tissue edema. [6]
Neuritis (/ nj ʊəˈr aɪ t ɪ s /), from the Greek νεῦρον), [1] is inflammation of a nerve [2] or the general inflammation of the peripheral nervous system.Inflammation, and frequently concomitant demyelination, [3] [4] [5] cause impaired transmission of neural signals and leads to aberrant nerve function.
The lateral rectus is the only muscle supplied by the abducens nerve (CN VI). The neuron cell bodies are located in the abducens nucleus in the pons.These neurons project axons as the abducens nerve which exit from the pontomedullary junction of the brainstem, travels through the cavernous sinus and enter the orbit through the superior orbital fissure.
Diabetic neuropathy can affect any peripheral nerves including sensory neurons, motor neurons, and the autonomic nervous system. Therefore, diabetic neuropathy has the potential to affect essentially any organ system and can cause a range of symptoms. There are several distinct syndromes based on the organ systems affected. [citation needed]
Composition and central connections of the spinal nerves; Pathways from the brain to the spinal cord; The meninges of the brain and medulla spinalis; The cerebrospinal fluid; The cranial nerves. The olfactory nerves; The optic nerve; The oculomotor nerve; The trochlear nerve; The trigeminal nerve; The abducens nerve; The facial nerve; The ...
The thickened and enhanced symptomatic nerve, as shown in recent MRI findings, might be indicative of the existence of structural nerve damage in RPON. [12] Therefore, neuropathy is suggested as the primary cause of RPON, [21] either induced by recurrent viral infections or immune-mediated inflammation. [22]
The cholinergic anti-inflammatory pathway provides a braking effect on the innate immune response which protects the body against the damage that can occur if a localized inflammatory response spreads beyond the local tissues, which results in toxicity or damage to the kidney, liver, lungs, and other organs. [5]